VE-cadherin cleavage by LasB protease from Pseudomonas aeruginosa facilitates type III secretion system toxicity in endothelial cells.
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Pseudomonas aeruginosa Transmigrates at Epithelial Cell-Cell Junctions, Exploiting Sites of Cell Division and Senescent Cell ExtrusionExpanding Role of Type II Secretion in Bacterial Pathogenesis and BeyondChronic wound infections: the role of Pseudomonas aeruginosa and Staphylococcus aureus.Pseudomonas aeruginosa: breaking down barriers.Pseudomonas aeruginosa ExlA and Serratia marcescens ShlA trigger cadherin cleavage by promoting calcium influx and ADAM10 activation.Stiff Substrates Increase Inflammation-Induced Endothelial Monolayer Tension and Permeability.Pseudomonas aeruginosa renews its virulence factors.Campylobacter jejuni outer membrane vesicle-associated proteolytic activity promotes bacterial invasion by mediating cleavage of intestinal epithelial cell E-cadherin and occludin.Bacteria and endothelial cells: a toxic relationship.The contribution of Pseudomonas aeruginosa virulence factors and host factors in the establishment of urinary tract infections.Pseudomonas aeruginosa proteolytically alters the interleukin 22-dependent lung mucosal defense.Phenotype and toxicity of the recently discovered exlA-positive Pseudomonas aeruginosa strains collected worldwide.Pancreatic fistulae secondary to trypsinogen activation by Pseudomonas aeruginosa infection after pancreatoduodenectomy.Autophagy Inhibitor 3-Methyladenine Protects against Endothelial Cell Barrier Dysfunction in Acute Lung Injury.Pseudomonas aeruginosa Takes a Multi-Target Approach to Achieve Junction Breach.Importins α and β signaling mediates endothelial cell inflammation and barrier disruption.CLIQ-BID: A method to quantify bacteria-induced damage to eukaryotic cells by automated live-imaging of bright nuclei.Endothelial cell SHP-2 negatively regulates neutrophil adhesion and promotes transmigration by enhancing ICAM-1-VE-cadherin interaction.Mechanisms and Targeted Therapies for Pseudomonas aeruginosa Lung Infection.in Chronic Lung Infections: How to Adapt Within the Host?Impact of quorum sensing signaling molecules on adhesion and inflammatory markers in endothelial cellsIncreased Virulence of Bloodstream Over Peripheral Isolates of Identified Through Post-transcriptional Regulation of Virulence Factors
P2860
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P2860
VE-cadherin cleavage by LasB protease from Pseudomonas aeruginosa facilitates type III secretion system toxicity in endothelial cells.
description
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VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells.
@ast
VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells.
@en
type
label
VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells.
@ast
VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells.
@en
prefLabel
VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells.
@ast
VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells.
@en
P2093
P2860
P1433
P1476
VE-cadherin cleavage by LasB p ...... toxicity in endothelial cells
@en
P2093
Eric Faudry
Guillaume Golovkine
Romé Voulhoux
Stéphanie Bouillot
P2860
P304
P356
10.1371/JOURNAL.PPAT.1003939
P577
2014-03-13T00:00:00Z