Reduction in reactive oxygen species production by mitochondria from elderly subjects with normal and impaired glucose tolerance - Wikidata - awgldk - TriplyDB

Reduction in reactive oxygen species production by mitochondria from elderly subjects with normal and impaired glucose tolerance

Reduction in reactive oxygen species production by mitochondria from elderly subjects with normal and impaired glucose tolerance

http://www.wikidata.org/entity/Q35123509

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Mitochondrial Dysfunction in Alzheimer's Disease and the Rationale for Bioenergetics Based Therapies Mitochondrial Quality Control and Muscle Mass Maintenance Mitochondrial aging and age-related dysfunction of mitochondria Impact of oxidative stress on exercising skeletal muscle Mitochondrial and skeletal muscle health with advancing age Oxidative stress in aging: advances in proteomic approaches Redox signaling in skeletal muscle: role of aging and exercise Oxygen consumption and usage during physical exercise: the balance between oxidative stress and ROS-dependent adaptive signaling Low abundance of the matrix arm of complex I in mitochondria predicts longevity in mice.Loss of Pgc-1α expression in aging mouse muscle potentiates glucose intolerance and systemic inflammation Diabetes and cardiovascular disease in older adults: current status and future directions.Protein carbonylation and heat shock proteins in human skeletal muscle: relationships to age and sarcopenia.Elevated muscle TLR4 expression and metabolic endotoxemia in human aging.Combined training enhances skeletal muscle mitochondrial oxidative capacity independent of age Defects in mitochondrial efficiency and H2O2 emissions in obese women are restored to a lean phenotype with aerobic exercise training.Immune aging, dysmetabolism, and inflammation in neurological diseases.Redox regulation of muscle adaptations to contractile activity and aging.Skeletal muscle mitochondria and aging: a review.The interrelationship between air temperature and humidity as applied locally to the skin: the resultant response on skin temperature and blood flow with age differences The Ups and Downs of Insulin Resistance and Type 2 Diabetes: Lessons from Genomic Analyses in Humans Skeletal muscle aging and the mitochondrion Endurance training increases the efficiency of rat skeletal muscle mitochondria.Genetic disruption of SOD1 gene causes glucose intolerance and impairs β-cell function.Pioglitazone is equally effective for diabetes prevention in older versus younger adults with impaired glucose tolerance.Markers of human skeletal muscle mitochondrial biogenesis and quality control: effects of age and aerobic exercise training.Skeletal muscle aging: influence of oxidative stress and physical exercise.Mitochondria in metabolic disease: getting clues from proteomic studies.Mitohormesis in muscle cells: a morphological, molecular, and proteomic approach.Exercise improves mitochondrial and redox-regulated stress responses in the elderly: better late than never!Mitochondria, muscle health, and exercise with advancing age.Radical Oxygen Species, Exercise and Aging: An Update.Autophagy as a Potential Target for Sarcopenia.L-Lysine suppresses myofibrillar protein degradation and autophagy in skeletal muscles of senescence-accelerated mouse prone 8.Mechanisms of muscular electrophysiological and mitochondrial dysfunction following exposure to malathion, an organophosphorus pesticide.Skeletal muscle mitochondrial H2 O2 emission increases with immobilization and decreases after aerobic training in young and older men.PGC-1α affects aging-related changes in muscle and motor function by modulating specific exercise-mediated changes in old mice.A novel method for determining human ex vivo submaximal skeletal muscle mitochondrial function.Properties of the vastus lateralis muscle in relation to age and physiological function in master cyclists aged 55-79 years.Strength training improves muscle aerobic capacity and glucose tolerance in elderly.Cultured senescent myoblasts derived from human vastus lateralis exhibit normal mitochondrial ATP synthesis capacities with correlating concomitant ROS production while whole cell ATP production is decreased.

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P2860

Reduction in reactive oxygen species production by mitochondria from elderly subjects with normal and impaired glucose tolerance

http://www.wikidata.org/entity/Q35123509

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2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Reduction in reactive oxygen s ...... and impaired glucose tolerance

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Reduction in reactive oxygen s ...... and impaired glucose tolerance

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Reduction in reactive oxygen s ...... and impaired glucose tolerance

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Reduction in reactive oxygen s ...... and impaired glucose tolerance

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Arlan Richardson

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Benjamin P Bowen

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Joaquin Joya-Galeana

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Jose de Jesus Garduno-Garcia

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Lawrence Mandarino

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Marjorie Molina-Carrion

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Muhammad Abdul-Ghani

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Natalie Lefort

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Nicolas Musi

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Raweewan Lertwattanarak

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P2860

Reduction in the Incidence of Type 2 Diabetes with Lifestyle Intervention or Metformin

Diagnosis and Classification of Diabetes Mellitus

Correction of hyperglycemia with phlorizin normalizes tissue sensitivity to insulin in diabetic rats

Peroxisome proliferator-activated receptor gamma coactivator 1 coactivators, energy homeostasis, and metabolism

Mortality in adults with and without diabetes in a national cohort of the U.S. population, 1971-1993

Mitochondrial damage by the "pro-oxidant" peroxisomal proliferator clofibrate

Dysfunction of mitochondria in human skeletal muscle in type 2 diabetes

Mitochondrial dysfunction in the elderly: possible role in insulin resistance

Reactive oxygen species have a causal role in multiple forms of insulin resistance

Reactive oxygen in skeletal muscle. I. Intracellular oxidant kinetics and fatigue in vitro.

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2051-2060

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10.2337/DB11-0121

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8

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60

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Ralph A DeFronzo

Holly Van Remmen

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2011-06-15T00:00:00Z

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51223035

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