Caenorhabditis elegans as a model for stem cell biology.
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Caenorhabditis elegans as a model for cancer researchConcise review: The plasticity of stem cell niches: a general property behind tissue homeostasis and repair.Alternative Polyadenylation Directs Tissue-Specific miRNA Targeting in Caenorhabditis elegans Somatic Tissues.Pluripotent cells will not dosage compensate.Basic Caenorhabditis elegans methods: synchronization and observation.Transdifferentiation and remodeling of post-embryonic C. elegans cells by a single transcription factorControl of stem cell self-renewal and differentiation by the heterochronic genes and the cellular asymmetry machinery in Caenorhabditis elegans.Targeting Homologous Recombination in Notch-Driven C. elegans Stem Cell and Human Tumors.Balancing up and downregulation of the C. elegans X chromosomes.Identification of Wnt Pathway Target Genes Regulating the Division and Differentiation of Larval Seam Cells and Vulval Precursor Cells in Caenorhabditis elegansThe C. elegans embryonic fate specification factor EGL-18 (GATA) is reutilized downstream of Wnt signaling to maintain a population of larval progenitor cells.Asymmetric Wnt Pathway Signaling Facilitates Stem Cell-Like Divisions via the Nonreceptor Tyrosine Kinase FRK-1 in Caenorhabditis elegans.Essential role for Notch signaling in restricting developmental plasticity.C. elegans GATA factors EGL-18 and ELT-6 function downstream of Wnt signaling to maintain the progenitor fate during larval asymmetric divisions of the seam cellsThe Paired-box protein PAX-3 regulates the choice between lateral and ventral epidermal cell fates in C. elegans.The NF-Y complex negatively regulates Caenorhabditis elegans tbx-2 expression.The onset of C. elegans dosage compensation is linked to the loss of developmental plasticity.Insulin and germline proliferation in Caenorhabditis elegans.New insights into mechanisms of stem cell daughter fate determination in regenerative tissues.Therapeutic Application of Adult Stem Cells in the Heart.Finding a niche for seam cells?CED-3 caspase acts with miRNAs to regulate non-apoptotic gene expression dynamics for robust development in C. elegans.The ortholog of the human proto-oncogene ROS1 is required for epithelial development in C. elegans.kin-19/casein kinase Iα has dual functions in regulating asymmetric division and terminal differentiation in C. elegans epidermal stem cells.Function following form: functional differentiation of mammary epithelial cells requires laminin-induced polarization of PI3-kinase.Live imaging of cellular dynamics during Caenorhabditis elegans postembryonic development.Coupled Caspase and N-End Rule Ligase Activities Allow Recognition and Degradation of Pluripotency Factor LIN-28 during Non-Apoptotic Development.Analysis of the C. elegans Germline Stem Cell Pool.
P2860
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P2860
Caenorhabditis elegans as a model for stem cell biology.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Caenorhabditis elegans as a model for stem cell biology.
@ast
Caenorhabditis elegans as a model for stem cell biology.
@en
type
label
Caenorhabditis elegans as a model for stem cell biology.
@ast
Caenorhabditis elegans as a model for stem cell biology.
@en
prefLabel
Caenorhabditis elegans as a model for stem cell biology.
@ast
Caenorhabditis elegans as a model for stem cell biology.
@en
P2093
P2860
P356
P1476
Caenorhabditis elegans as a model for stem cell biology.
@en
P2093
Joel H Rothman
Misty R Riddle
Nareg J V Djabrayan
P2860
P304
P356
10.1002/DVDY.22296
P577
2010-05-01T00:00:00Z