Extensive aspartoacylase expression in the rat central nervous system.
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Expression and localization of myosin-1d in the developing nervous systemLocalisation of N-acetylaspartate in oligodendrocytes/myelinN-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylationA guide to the metabolic pathways and function of metabolites observed in human brain 1H magnetic resonance spectra.Increasing N-acetylaspartate in the Brain during Postnatal Myelination Does Not Cause the CNS Pathologies of Canavan Disease.Acetate supplementation induces growth arrest of NG2/PDGFRα-positive oligodendroglioma-derived tumor-initiating cellsPharmaceutical integrated stress response enhancement protects oligodendrocytes and provides a potential multiple sclerosis therapeuticAcetate supplementation as a means of inducing glioblastoma stem-like cell growth arrestN-acetylaspartate catabolism determines cytosolic acetyl-CoA levels and histone acetylation in brown adipocytesCharacterization of glucose-related metabolic pathways in differentiated rat oligodendrocyte lineage cellsN-acetylaspartate (NAA) and N-acetylaspartylglutamate (NAAG) promote growth and inhibit differentiation of glioma stem-like cellsDecreased NAA in gray matter is correlated with decreased availability of acetate in white matter in postmortem multiple sclerosis cortex.A thematic review of scientific and family interests in Canavan Disease: where are the developmentalists?Adapting brain metabolism to myelination and long-range signal transduction.Short-term cuprizone feeding verifies N-acetylaspartate quantification as a marker of neurodegeneration.Oligodendrocytes: Development, Physiology and Glucose Metabolism.The neuronal metabolite NAA regulates histone H3 methylation in oligodendrocytes and myelin lipid composition.Triacetin-based acetate supplementation as a chemotherapeutic adjuvant therapy in glioma.Astroglial redistribution of aquaporin 4 during spongy degeneration in a Canavan disease mouse model.Uncoupling N-acetylaspartate from brain pathology: implications for Canavan disease gene therapy.Acetyl-CoA production from pyruvate is not necessary for preservation of myelin.Combining Double Fluorescence In Situ Hybridization with Immunolabelling for Detection of the Expression of Three Genes in Mouse Brain Sections.
P2860
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P2860
Extensive aspartoacylase expression in the rat central nervous system.
description
2011 nî lūn-bûn
@nan
2011 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Extensive aspartoacylase expression in the rat central nervous system.
@ast
Extensive aspartoacylase expression in the rat central nervous system.
@en
type
label
Extensive aspartoacylase expression in the rat central nervous system.
@ast
Extensive aspartoacylase expression in the rat central nervous system.
@en
prefLabel
Extensive aspartoacylase expression in the rat central nervous system.
@ast
Extensive aspartoacylase expression in the rat central nervous system.
@en
P2093
P2860
P356
P1433
P1476
Extensive aspartoacylase expression in the rat central nervous system.
@en
P2093
Aryan M A Namboodiri
David M Jacobowitz
James Y Garbern
John R Moffett
Peethambaran Arun
P2860
P304
P356
10.1002/GLIA.21186
P577
2011-05-19T00:00:00Z