Glucocorticoids regulate tristetraprolin synthesis and posttranscriptionally regulate tumor necrosis factor alpha inflammatory signaling. - Wikidata - awgldk - TriplyDB

Glucocorticoids regulate tristetraprolin synthesis and posttranscriptionally regulate tumor necrosis factor alpha inflammatory signaling.

Glucocorticoids regulate tristetraprolin synthesis and posttranscriptionally regulate tumor necrosis factor alpha inflammatory signaling.

http://www.wikidata.org/entity/Q35131624

about

The roles of TTP and BRF proteins in regulated mRNA decay Glucocorticoid receptor signaling in health and disease One hormone, two actions: anti- and pro-inflammatory effects of glucocorticoids Endothelial Response to Glucocorticoids in Inflammatory Diseases The control of inflammation via the phosphorylation and dephosphorylation of tristetraprolin: a tale of two phosphatases ZFP36L1 is regulated by growth factors and cytokines in keratinocytes and influences their VEGF production Phosphorylation site analysis of the anti-inflammatory and mRNA-destabilizing protein tristetraprolin.Crosstalk in inflammation: the interplay of glucocorticoid receptor-based mechanisms and kinases and phosphatases Dexamethasone restrains ongoing expression of interleukin-23p19 in peripheral blood-derived human macrophages.Mammary differentiation induces expression of Tristetraprolin, a tumor suppressor AU-rich mRNA-binding protein.The human glucocorticoid receptor as an RNA-binding protein: global analysis of glucocorticoid receptor-associated transcripts and identification of a target RNA motif.Glucocorticosteroids: current and future directions.Biochemical basis of asthma therapy.Green tea increases anti-inflammatory tristetraprolin and decreases pro-inflammatory tumor necrosis factor mRNA levels in rats.Negative Feed-forward Control of Tumor Necrosis Factor (TNF) by Tristetraprolin (ZFP36) Is Limited by the Mitogen-activated Protein Kinase Phosphatase, Dual-specificity Phosphatase 1 (DUSP1): IMPLICATIONS FOR REGULATION BY GLUCOCORTICOIDS.Muscle transcriptome response to ACTH administration in a free-ranging marine mammal.Regulation of cytoplasmic mRNA decay.The role of tristetraprolin in cancer and inflammation.Dexamethasone Induces FcγRIIb Expression in RBL-2H3 Cells Nuclear tristetraprolin acts as a corepressor of multiple steroid nuclear receptors in breast cancer cells.Role of the RNA-binding protein tristetraprolin in glucocorticoid-mediated gene regulation.The five Rs of glucocorticoid action during inflammation: ready, reinforce, repress, resolve, and restore.Mechanism of upregulation of the inhibitory regulator, src-like adaptor protein (SLAP), by glucocorticoids in mast cells.TIS11 family proteins and their roles in posttranscriptional gene regulation.mRNA-binding protein ZFP36 is expressed in atherosclerotic lesions and reduces inflammation in aortic endothelial cells Coordinated post-transcriptional regulation of the chemokine system: messages from CCL2 Tristetraprolin: roles in cancer and senescence.Posttranscriptional Gene Regulation: Novel Pathways for Glucocorticoids' Anti-inflammatory Action.Glucocorticoid sensitivity in health and disease.Post-transcriptional regulation of cytokine mRNA controls the initiation and resolution of inflammation.Pharmacological basis and scientific rationale underlying the targeted use of inhaled corticosteroid/long-acting β2-adrenergic agonist combinations in chronic obstructive pulmonary disease treatment.Glucocorticoid receptors: finding the middle ground.Understanding how long-acting β2 -adrenoceptor agonists enhance the clinical efficacy of inhaled corticosteroids in asthma - an update.Glucocorticoid and cytokine crosstalk: Feedback, feedforward, and co-regulatory interactions determine repression or resistance.Regulation of mRNA stability by CCCH-type zinc-finger proteins in immune cells.Mouse models of severe asthma: Understanding the mechanisms of steroid resistance, tissue remodelling and disease exacerbation.Anti-inflammatory effects of selective glucocorticoid receptor modulators are partially dependent on up-regulation of dual specificity phosphatase 1 Casein kinase 2 regulates the mRNA-destabilizing activity of tristetraprolin.Krüppel-like Factor 13 Is a Major Mediator of Glucocorticoid Receptor Signaling in Cardiomyocytes and Protects These Cells from DNA Damage and Death.Transcriptomic analysis of skeletal muscle from beef cattle exposed to illicit schedules containing dexamethasone: identification of new candidate biomarkers and their validation using samples from a field monitoring trial.

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Glucocorticoids regulate tristetraprolin synthesis and posttranscriptionally regulate tumor necrosis factor alpha inflammatory signaling.

http://www.wikidata.org/entity/Q35131624

description

2006 nî lūn-bûn

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2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Glucocorticoids regulate trist ...... alpha inflammatory signaling.

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Glucocorticoids regulate trist ...... alpha inflammatory signaling.

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type

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Glucocorticoids regulate trist ...... alpha inflammatory signaling.

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Glucocorticoids regulate trist ...... alpha inflammatory signaling.

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Glucocorticoids regulate trist ...... alpha inflammatory signaling.

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Glucocorticoids regulate trist ...... alpha inflammatory signaling.

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Molecular and Cellular Biology

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Glucocorticoids regulate trist ...... r alpha inflammatory signaling

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P2093

Kathleen Smoak

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P2860

Evidence that tristetraprolin binds to AU-rich elements and promotes the deadenylation and destabilization of tumor necrosis factor alpha mRNA.

Feedback inhibition of macrophage tumor necrosis factor-alpha production by tristetraprolin

Dexamethasone causes sustained expression of mitogen-activated protein kinase (MAPK) phosphatase 1 and phosphatase-mediated inhibition of MAPK p38.

Tristetraprolin and its family members can promote the cell-free deadenylation of AU-rich element-containing mRNAs by poly(A) ribonuclease

The role of mRNA turnover in the regulation of tristetraprolin expression: evidence for an extracellular signal-regulated kinase-specific, AU-rich element-dependent, autoregulatory pathway

Tristetraprolin down-regulates IL-2 gene expression through AU-rich element-mediated mRNA decay

Analysis of the function, expression, and subcellular distribution of human tristetraprolin

Rapid insulin-stimulated accumulation of an mRNA encoding a proline-rich protein

A growth factor-inducible nuclear protein with a novel cysteine/histidine repetitive sequence

A pathogenetic role for TNF alpha in the syndrome of cachexia, arthritis, and autoimmunity resulting from tristetraprolin (TTP) deficiency

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9126-9135

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scholarly article

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10.1128/MCB.00679-06

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23

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26

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John A Cidlowski

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2006-09-18T00:00:00Z

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6811030

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1636823

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