CD8α dendritic cells drive establishment of HSV-1 latency
about
Immunological aspects of acute and recurrent herpes simplex keratitisGenital Herpes: Insights into Sexually Transmitted Infectious DiseaseRole of CD8+ T cells and lymphoid dendritic cells in protection from ocular herpes simplex virus 1 challenge in immunized mice.CD8+ T Cells Play a Bystander Role in Mice Latently Infected with Herpes Simplex Virus 1.Interrelationship of Primary Virus Replication, Level of Latency, and Time to Reactivation in the Trigeminal Ganglia of Latently Infected Mice.Dendritic cells, macrophages, NK and CD8+ T lymphocytes play pivotal roles in controlling HSV-1 in the trigeminal ganglia by producing IL1-beta, iNOS and granzyme B.Immune response of T cells during herpes simplex virus type 1 (HSV-1) infection.Dok-1 and Dok-2 Are Required To Maintain Herpes Simplex Virus 1-Specific CD8+ T Cells in a Murine Model of Ocular Infection.Coregulatory interactions among CD8α dendritic cells, the latency-associated transcript, and programmed death 1 contribute to higher levels of herpes simplex virus 1 latency.Batf3 deficiency is not critical for the generation of CD8α⁺ dendritic cells.
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P2860
CD8α dendritic cells drive establishment of HSV-1 latency
description
2014 nî lūn-bûn
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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2014 թվականի ապրիլին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
CD8α dendritic cells drive establishment of HSV-1 latency
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CD8α dendritic cells drive establishment of HSV-1 latency
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type
label
CD8α dendritic cells drive establishment of HSV-1 latency
@ast
CD8α dendritic cells drive establishment of HSV-1 latency
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prefLabel
CD8α dendritic cells drive establishment of HSV-1 latency
@ast
CD8α dendritic cells drive establishment of HSV-1 latency
@en
P2093
P2860
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CD8α dendritic cells drive establishment of HSV-1 latency
@en
P2093
Behrooz G Sharifi
Bindu Konda
Clinton Jones
Homayon Ghiasi
Kevin R Mott
Mandana Zandian
Sariah J Allen
Steven L Wechsler
Terrence Town
P2860
P304
P356
10.1371/JOURNAL.PONE.0093444
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P577
2014-04-02T00:00:00Z