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Are we getting closer to the treatment of rabies?IL-33 signaling is essential to attenuate viral-induced encephalitis development by downregulating iNOS expression in the central nervous systemImmunobiology of congenital cytomegalovirus infection of the central nervous system—the murine cytomegalovirus modelTregs Modulate Lymphocyte Proliferation, Activation, and Resident-Memory T-Cell Accumulation within the Brain during MCMV InfectionAlpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survivalPathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral EvasionCellular Inflammatory Response to Flaviviruses in the Central Nervous System of a Primate HostVaccines for Venezuelan equine encephalitisJapanese Encephalitis Virus Induce Immuno-Competency in Neural Stem/Progenitor CellsFast type I interferon response protects astrocytes from flavivirus infection and virus-induced cytopathic effectsEvaluation of Possible Consequences of Zika Virus Infection in the Developing Nervous SystemIFNβ Protects Neurons from Damage in a Murine Model of HIV-1 Associated Brain InjuryRegulation of SIV mac 239 basal long terminal repeat activity and viral replication in macrophages: functional roles of two CCAAT/enhancer-binding protein beta sites in activation and interferon beta-mediated suppression.Virus-specific and bystander CD8 T cells recruited during virus-induced encephalomyelitisGamma interferon-dependent, noncytolytic clearance of sindbis virus infection from neurons in vitroSilencing of neurotropic flavivirus replication in the central nervous system by combining multiple microRNA target insertions in two distinct viral genome regionsT cells facilitate recovery from Venezuelan equine encephalitis virus-induced encephalomyelitis in the absence of antibody.Human neuronal cells possess functional cytoplasmic and TLR-mediated innate immune pathways influenced by phosphatidylinositol-3 kinase signaling.Recovery from viral encephalomyelitis: immune-mediated noncytolytic virus clearance from neuronsPoly-thymidine oligonucleotides mediate activation of murine glial cells primarily through TLR7, not TLR8.Defining the chemokine basis for leukocyte recruitment during viral encephalitis.CD8+ T lymphocytes control murine cytomegalovirus replication in the central nervous system of newborn animals.Enhancement of blood-brain barrier permeability is required for intravenously administered virus neutralizing antibodies to clear an established rabies virus infection from the brain and prevent the development of rabies in mice.Early emergence and selection of a SIV-LTR C/EBP site variant in SIV-infected macaques that increases virus infectivityGlucocorticoid treatment of MCMV infected newborn mice attenuates CNS inflammation and limits deficits in cerebellar development.Factors supporting intrathecal humoral responses following viral encephalomyelitis.MicroRNA regulation of IFN-beta protein expression: rapid and sensitive modulation of the innate immune response.Altered central nervous system gene expression caused by congenitally acquired persistent infection with lymphocytic choriomeningitis virus.Inhibition of interferon-gamma signaling in oligodendroglia delays coronavirus clearance without altering demyelination.TLR7 and TLR9 trigger distinct neuroinflammatory responses in the CNSThe role of the blood-brain barrier during Venezuelan equine encephalitis virus infection.The critical role of antigen-presentation-induced cytokine crosstalk in the central nervous system in multiple sclerosis and experimental autoimmune encephalomyelitis.A preliminary randomized double blind placebo-controlled trial of intravenous immunoglobulin for Japanese encephalitis in Nepal.Transplacental murine cytomegalovirus infection in the brain of SCID miceSynergistic roles of antibody and interferon in noncytolytic clearance of Sindbis virus from different regions of the central nervous system.Immune-mediated loss of transgene expression from virally transduced brain cells is irreversible, mediated by IFNγ, perforin, and TNFα, and due to the elimination of transduced cellsDistinct Immune Responses in Resistant and Susceptible Strains of Mice during Neurovirulent Alphavirus Encephalomyelitis.Immunological thresholds in neurological gene therapy: highly efficient elimination of transduced cells might be related to the specific formation of immunological synapses between T cells and virus-infected brain cells.Immunotherapeutic relief from persistent infections and amyloid disorders.NKG2D receptor signaling enhances cytolytic activity by virus-specific CD8+ T cells: evidence for a protective role in virus-induced encephalitis.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Immune responses to RNA-virus infections of the CNS.
@ast
Immune responses to RNA-virus infections of the CNS.
@en
type
label
Immune responses to RNA-virus infections of the CNS.
@ast
Immune responses to RNA-virus infections of the CNS.
@en
prefLabel
Immune responses to RNA-virus infections of the CNS.
@ast
Immune responses to RNA-virus infections of the CNS.
@en
P356
P1476
Immune responses to RNA-virus infections of the CNS.
@en
P304
P356
10.1038/NRI1105
P577
2003-06-01T00:00:00Z