LIGHT/TNFSF14 increases osteoclastogenesis and decreases osteoblastogenesis in multiple myeloma-bone disease. - Wikidata - awgldk - TriplyDB

LIGHT/TNFSF14 increases osteoclastogenesis and decreases osteoblastogenesis in multiple myeloma-bone disease.

LIGHT/TNFSF14 increases osteoclastogenesis and decreases osteoblastogenesis in multiple myeloma-bone disease.

http://www.wikidata.org/entity/Q35149611

about

Signaling Interplay between Bone Marrow Adipose Tissue and Multiple Myeloma cells Bone Regeneration Induced by Bone Porcine Block with Bone Marrow Stromal Stem Cells in a Minipig Model of Mandibular "Critical Size" Defect.Role of LIGHT in the pathogenesis of joint destruction in rheumatoid arthritis.Bone-immune cell crosstalk: bone diseases.LIGHT (TNFSF14) Increases the Survival and Proliferation of Human Bone Marrow-Derived Mesenchymal Stem Cells Clinical prognostic significance and pro-metastatic activity of RANK/RANKL via the AKT pathway in endometrial cancer Immunotherapeutic targeting of LIGHT/LTβR/HVEM pathway fully recapitulates the reduced cytotoxic phenotype of LIGHT-deficient T cells.Non-Canonical (RANKL-Independent) Pathways of Osteoclast Differentiation and Their Role in Musculoskeletal Diseases.Adipose Derived-Mesenchymal Stem Cells Viability and Differentiating Features for Orthopaedic Reparative Applications: Banking of Adipose Tissue.Emerging treatment approaches for myeloma-related bone disease.TGF-β2-induced ANGPTL4 expression promotes tumor progression and osteoclast differentiation in giant cell tumor of bone.Inflammation induces osteoclast differentiation from peripheral mononuclear cells in chronic kidney disease patients: crosstalk between the immune and bone systems.Immunology of Gut-Bone Signaling.Monoclonal antibodies for treating osteoporosis.Shining LIGHT on the metabolic role of the cytokine TNFSF14 and the implications on hepatic IL-6 production.Pathogenesis of bone disease in multiple myeloma: from bench to bedside.Impairment of Bone Remodeling in LIGHT/TNFSF14-Deficient Mice.Deficient invariant natural killer T cells had impaired regulation on osteoclastogenesis in myeloma bone disease.LIGHT/TNFSF14 as a New Biomarker of Bone Disease in Multiple Myeloma Patients Experiencing Therapeutic Regimens Osteoclast Immunosuppressive Effects in Multiple Myeloma: Role of Programmed Cell Death Ligand 1

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P2860

LIGHT/TNFSF14 increases osteoclastogenesis and decreases osteoblastogenesis in multiple myeloma-bone disease.

http://www.wikidata.org/entity/Q35149611

description

2014 nî lūn-bûn

@nan

2014 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease.

@ast

LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease.

@en

type

label

LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease.

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LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease.

@en

prefLabel

LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease.

@ast

LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease.

@en

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ONCOTARGET.2633

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P1476

LIGHT/TNFSF14 increases osteoc ...... multiple myeloma-bone disease

@en

P2093

Adriana Di Benedetto

http://www.w3.org/2001/XMLSchema#string

Angela Oranger

http://www.w3.org/2001/XMLSchema#string

Anna Mestice

http://www.w3.org/2001/XMLSchema#string

Anna Napoli

http://www.w3.org/2001/XMLSchema#string

Giorgina Specchia

http://www.w3.org/2001/XMLSchema#string

Giorgio Mori

http://www.w3.org/2001/XMLSchema#string

Giuseppe Ingravallo

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Grazia Taurino

http://www.w3.org/2001/XMLSchema#string

Isabella Gigante

http://www.w3.org/2001/XMLSchema#string

Maria Felicia Faienza

http://www.w3.org/2001/XMLSchema#string

P2860

Herpesvirus entry mediator ligand (HVEM-L), a novel ligand for HVEM/TR2, stimulates proliferation of T cells and inhibits HT29 cell growth

The lymphotoxin-beta receptor is necessary and sufficient for LIGHT-mediated apoptosis of tumor cells

LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator

ATAR, a novel tumor necrosis factor receptor family member, signals through TRAF2 and TRAF5

Herpesvirus entry mediator, a member of the tumor necrosis factor receptor (TNFR) family, interacts with members of the TNFR-associated factor family and activates the transcription factors NF-kappaB and AP-1

LIGHT, a novel ligand for lymphotoxin beta receptor and TR2/HVEM induces apoptosis and suppresses in vivo tumor formation via gene transfer

Criteria for diagnosis, staging, risk stratification and response assessment of multiple myeloma

The regulation of T cell homeostasis and autoimmunity by T cell-derived LIGHT

TRANCE, a TNF family member, activates Akt/PKB through a signaling complex involving TRAF6 and c-Src

Constitutive expression of LIGHT on T cells leads to lymphocyte activation, inflammation, and tissue destruction

P304

12950-12967

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scholarly article

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10.18632/ONCOTARGET.2633

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24

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5

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Giacomina Brunetti

Graziana Colaianni

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2014-12-01T00:00:00Z

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269104524

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25460501

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4350341

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