IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis. - Wikidata - awgldk - TriplyDB

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

http://www.wikidata.org/entity/Q35153201

about

Animal models of anti-neutrophil cytoplasmic antibody-associated vasculitis The neutrophil in antineutrophil cytoplasmic autoantibody-associated vasculitis Monocyte recruitment by HLA IgG-activated endothelium: the relationship between IgG subclass and FcγRIIa polymorphisms.Crystal structure of Streptococcus pyogenes EndoS, an immunomodulatory endoglycosidase specific for human IgG antibodies Pathophysiology of ANCA-associated small vessel vasculitis Antineutrophilic cytoplasmic antibody-associated vasculitis with hypocomplementemia has a higher incidence of serious organ damage and a poor prognosis.Study of the IgG endoglycosidase EndoS in group A streptococcal phagocyte resistance and virulence.Influence of variable domain glycosylation on anti-neutrophil cytoplasmic autoantibodies and anti-glomerular basement membrane autoantibodies.EndoS reduces the pathogenicity of anti-mCOL7 IgG through reduced binding of immune complexes to neutrophils.Bacterial glycosidases in pathogenesis and glycoengineering.EndoS and EndoS2 hydrolyze Fc-glycans on therapeutic antibodies with different glycoform selectivity and can be used for rapid quantification of high-mannose glycans Neutrophil serine proteases promote IL-1β generation and injury in necrotizing crescentic glomerulonephritis.Pro-inflammatory pattern of IgG1 Fc glycosylation in multiple sclerosis cerebrospinal fluid.CP40 from Corynebacterium pseudotuberculosis is an endo-β-N-acetylglucosaminidase.Endothelium-neutrophil interactions in ANCA-associated diseases IgG glycan hydrolysis by EndoS inhibits experimental autoimmune encephalomyelitis Enzymatic deglycosylation converts pathogenic neuromyelitis optica anti-aquaporin-4 immunoglobulin G into therapeutic antibody.Glycosylation Profile of IgG in Moderate Kidney Dysfunction.Clinical presentation, pathogenesis, diagnosis, and treatment of epidermolysis bullosa acquisita EndoS2 is a unique and conserved enzyme of serotype M49 group A Streptococcus that hydrolyses N-linked glycans on IgG and α1-acid glycoprotein.The Association Between Glycosylation of Immunoglobulin G and Hypertension: A Multiple Ethnic Cross-Sectional Study Liquid-liquid diffusion crystallization improves the X-ray diffraction of EndoS, an endo-β-N-acetylglucosaminidase from Streptococcus pyogenes with activity on human IgG Pathogenesis of ANCA-associated vasculitis, an update.In vivo approaches to investigate ANCA-associated vasculitis: lessons and limitations.Pathogenesis of antineutrophil cytoplasmic autoantibody vasculitis.Pathogenesis of anti-neutrophil cytoplasmic autoantibody (ANCA)-associated vasculitis.T-helper cells as new players in ANCA-associated vasculitides.How anti-neutrophil cytoplasmic autoantibodies activate neutrophils.Pathogenesis of antineutrophil cytoplasmic autoantibody-associated small-vessel vasculitis.The carbohydrate switch between pathogenic and immunosuppressive antigen-specific antibodies.B cell-mediated pathogenesis of ANCA-mediated vasculitis.Pathogenesis of antineutrophil cytoplasmic autoantibody-mediated disease.Discovering potential drug-targets for personalized treatment of autoimmune disorders - what we learn from epidermolysis bullosa acquisita.EndoSd: an IgG glycan hydrolyzing enzyme in Streptococcus dysgalactiae subspecies dysgalactiae.Antibody Glycosylation Predicts Relapse in Autoimmune Vasculitis Toward Clinical use of the IgG Specific Enzymes IdeS and EndoS against Antibody-Mediated Diseases.Galactosylation and Sialylation Levels of IgG Predict Relapse in Patients With PR3-ANCA Associated Vasculitis.Characterization of novel endo-β-N-acetylglucosaminidases from Sphingobacterium species, Beauveria bassiana and Cordyceps militaris that specifically hydrolyze fucose-containing oligosaccharides and human IgG.Towards precision medicine in ANCA-associated vasculitis.Pathogenetic and Clinical Aspects of Anti-Neutrophil Cytoplasmic Autoantibody-Associated Vasculitides.

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P2860

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

http://www.wikidata.org/entity/Q35153201

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

@ast

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

@en

type

label

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

@ast

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

@en

prefLabel

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

@ast

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.

@en

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P1433

Journal of the American Society of Nephrology

P1476

IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis

@en

P2093

Arjen H Petersen

http://www.w3.org/2001/XMLSchema#string

Betty S van der Veen

http://www.w3.org/2001/XMLSchema#string

Coen A Stegeman

http://www.w3.org/2001/XMLSchema#string

Mirjan M van Timmeren

http://www.w3.org/2001/XMLSchema#string

P2860

Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice

Experimental Autoimmune Vasculitis

EndoS, a novel secreted protein from Streptococcus pyogenes with endoglycosidase activity on human IgG

Human IgG/Fc gamma R interactions are modulated by streptococcal IgG glycan hydrolysis

Severe impairment in early host defense against Candida albicans in mice deficient in myeloperoxidase

Fcgamma receptors as regulators of immune responses

IgG glycan hydrolysis by a bacterial enzyme as a therapy against autoimmune conditions.

Occurrence of autoantibodies to human leucocyte elastase in Wegener's granulomatosis and other inflammatory disorders.

Anti-neutrophil cytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygen radicals in vitro

Antiproteinase 3- and antimyeloperoxidase-associated vasculitis.

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1103-1114

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scholarly article

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10.1681/ASN.2009090984

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7

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21

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Thomas Hellmark

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2010-05-06T00:00:00Z

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20448018

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glomerulonephritis

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3152232

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