Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli - Wikidata - awgldk - TriplyDB

Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

http://www.wikidata.org/entity/Q35157197

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Hepatocytes: a key cell type for innate immunity Chronic IL-6 Administration Desensitizes IL-6 Response in Liver, Causes Hyperleptinemia and Aggravates Steatosis in Diet-Induced-Obese Mice Protective effects of silymarin against bisphenol A-induced hepatotoxicity in mouse liver.Interleukin-6 and rs1800796 locus single nucleotide polymorphisms in response to hypoxia/reoxygenation in hepatocytes.Hepatocyte-specific Smad7 deletion accelerates DEN-induced HCC via activation of STAT3 signaling in mice.Advances in liver regeneration.Hyperproduction of IL-6 caused by aberrant TDP-43 overexpression in high-fat diet-induced obese mice.Evaluation of Hepatotoxicity with Treatment Doses of Flucytosine and Amphotericin B for Invasive Fungal Infections.Hepatocytes as Immunological Agents.Hepatostat: Liver regeneration and normal liver tissue maintenance.Donor interleukin 6 gene polymorphisms predict the recurrence of hepatocellular carcinoma after liver transplantation.The role of the secretin/secretin receptor axis in inflammatory cholangiocyte communication via extracellular vesicles.Monocyte-endothelial cell interactions in the regulation of vascular sprouting and liver regeneration in mouse.Modeling Therapeutic Antibody-Small Molecule Drug-Drug Interactions Using a Three-Dimensional Perfusable Human Liver Coculture Platform.Baicalin in flavocoxid may act against hepatitis B virus via a pro-inflammatory pathway.Hepatocyte nuclear factors as possible C-reactive protein transcriptional inducer in the liver and white adipose tissue of rats with experimental chronic renal failure.Central role of T helper 17 cells in chronic hypoxia-induced pulmonary hypertension.Microscale 3D Liver Bioreactor for In Vitro Hepatotoxicity Testing under Perfusion Conditions.Effect of remote ischemic preconditioning in the expression of IL-6 and IL-10 in a rat model of liver ischemia-reperfusion injury.Kupffer cell-derived TNF-α promotes hepatocytes to produce CXCL1 and mobilize neutrophils in response to necrotic cells.

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

http://www.wikidata.org/entity/Q35157197

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2014 nî lūn-bûn

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2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի ապրիլին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Synthesis of IL-6 by hepatocytes is a normal response to common hepatic stimuli

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Anne Orr

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Callie A Norris

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Donna B Stolz

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George K Michalopoulos

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Josiah E Radder

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Meagan M Haynes

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Mu He

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Shirish Paranjpe

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Wendy M Mars

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William C Bowen

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P2860

Hepatocyte growth factor modulates interleukin-6 production in bone marrow derived macrophages: implications for inflammatory mediated diseases

Cell cycle effects resulting from inhibition of hepatocyte growth factor and its receptor c-Met in regenerating rat livers by RNA interference

Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8

Involvement of a NF-kappa B-like transcription factor in the activation of the interleukin-6 gene by inflammatory lymphokines

Preparation of isolated rat liver cells

Liver regeneration: from myth to mechanism.

Foxa1 and Foxa2 regulate bile duct development in mice.

Destabilization of interleukin-6 mRNA requires a putative RNA stem-loop structure, an AU-rich element, and the RNA-binding protein AUF1.

IL6 and the acute phase reaction.

Hepatocyte growth factor exerts its anti-inflammatory action by disrupting nuclear factor-kappaB signaling

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