Complement activation triggers metalloproteinases release inducing cervical remodeling and preterm birth in mice.
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The complement system and adverse pregnancy outcomesProgesterone is not the same as 17α-hydroxyprogesterone caproate: implications for obstetrical practiceThe Microbiome and Complement Activation: A Mechanistic Model for Preterm Birth.Invariant NKT Cell Activation Induces Late Preterm Birth That Is Attenuated by Rosiglitazone.Vaginal progesterone, but not 17α-hydroxyprogesterone caproate, has antiinflammatory effects at the murine maternal-fetal interface.An M1-like Macrophage Polarization in Decidual Tissue during Spontaneous Preterm Labor That Is Attenuated by Rosiglitazone Treatment.Human Chorionic Gonadotropin Has Anti-Inflammatory Effects at the Maternal-Fetal Interface and Prevents Endotoxin-Induced Preterm Birth, but Causes Dystocia and Fetal Compromise in Mice.Cervical remodeling/ripening at term and preterm delivery: the same mechanism initiated by different mediators and different effector cellsCervical strain determined by ultrasound elastography and its association with spontaneous preterm deliveryImmune cells in term and preterm laborFuture directions of clinical laboratory evaluation of pregnancy.Imaging of activated complement using ultrasmall superparamagnetic iron oxide particles (USPIO)--conjugated vectors: an in vivo in utero non-invasive method to predict placental insufficiency and abnormal fetal brain development.Loss of progesterone receptor-mediated actions induce preterm cellular and structural remodeling of the cervix and premature birth.Altered autophagic flux enhances inflammatory responses during inflammation-induced preterm labor.The role of complement in tumor growth15-epi-lipoxin A4 reduces the mortality of prematurely born pups in a mouse model of infection-induced preterm birth.Ultrasound-guided intrauterine injection of lipopolysaccharide as a novel model of preterm birth in the mousePlatelet-activating factor: a role in preterm delivery and an essential interaction with Toll-like receptor signaling in mice.Crosstalk between monocytes and myometrial smooth muscle in culture generates synergistic pro-inflammatory cytokine production and enhances myocyte contraction, with effects opposed by progesterone.Complement factors C1q, C3 and C5b-9 in the posterior sclera of guinea pigs with negative lens-defocused myopia.Hydrogen Sulfide Delays LPS-Induced Preterm Birth in Mice via Anti-Inflammatory PathwaysNotch Signaling in Inflammation-Induced Preterm Labor.The clinical significance of a positive Amnisure test in women with preterm labor and intact membranesDepletion of polymorphonuclear leukocytes has no effect on preterm delivery in a mouse model of Escherichia coli-induced laborComplement Activation in Placental Malaria.Vaginal progesterone vs. cervical cerclage for the prevention of preterm birth in women with a sonographic short cervix, previous preterm birth, and singleton gestation: a systematic review and indirect comparison metaanalysisImplementing Mass Cytometry at the Bedside to Study the Immunological Basis of Human Diseases: Distinctive Immune Features in Patients with a History of Term or Preterm Birth.Comparison of the mechanisms responsible for cervical remodeling in preterm and term labor.Maternal and fetal roles in bacterially induced preterm labor in the mouse.An imbalance between innate and adaptive immune cells at the maternal-fetal interface occurs prior to endotoxin-induced preterm birthProkineticin 1 induces a pro-inflammatory response in murine fetal membranes but does not induce preterm delivery.Objective Assessment of Cervical Stiffness after Administration of Misoprostol for Intrauterine Contraceptive Insertion.Matrix Metalloproteinase Expression in the Rat Myometrium During Pregnancy, Term Labor, and PostpartumRegulation of apoptosis and innate immune stimuli in inflammation-induced preterm labor.Decidual neutrophil infiltration is not required for preterm birth in a mouse model of infection-induced preterm labor.Complement in pregnancy: a delicate balance.Does an upper respiratory tract infection during pregnancy affect perinatal outcomes? A literature review.Prevention of preterm birth: harnessing science to address the global epidemic.The pathophysiology of human premature cervical remodeling resulting in spontaneous preterm birth: Where are we now?Inflammation and preterm birth.
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P2860
Complement activation triggers metalloproteinases release inducing cervical remodeling and preterm birth in mice.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Complement activation triggers ...... ing and preterm birth in mice.
@ast
Complement activation triggers ...... ing and preterm birth in mice.
@en
type
label
Complement activation triggers ...... ing and preterm birth in mice.
@ast
Complement activation triggers ...... ing and preterm birth in mice.
@en
prefLabel
Complement activation triggers ...... ing and preterm birth in mice.
@ast
Complement activation triggers ...... ing and preterm birth in mice.
@en
P2093
P2860
P1476
Complement activation triggers ...... ing and preterm birth in mice.
@en
P2093
Claus-Werner Franzke
Fengyuan Yang
Juan M Gonzalez
P2860
P304
P356
10.1016/J.AJPATH.2011.04.024
P407
P577
2011-06-12T00:00:00Z