Microglia/macrophages migrate through retinal epithelium barrier by a transcellular route in diabetic retinopathy: role of PKCζ in the Goto Kakizaki rat model. - Wikidata - awgldk - TriplyDB

Microglia/macrophages migrate through retinal epithelium barrier by a transcellular route in diabetic retinopathy: role of PKCζ in the Goto Kakizaki rat model.

Microglia/macrophages migrate through retinal epithelium barrier by a transcellular route in diabetic retinopathy: role of PKCζ in the Goto Kakizaki rat model.

http://www.wikidata.org/entity/Q35168171

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Cellular Barriers after Extravasation: Leukocyte Interactions with Polarized Epithelia in the Inflamed Tissue Contribution of microglia-mediated neuroinflammation to retinal degenerative diseases Diabetic Macular Edema Pathophysiology: Vasogenic versus Inflammatory Adaptive Müller cell responses to microglial activation mediate neuroprotection and coordinate inflammation in the retina BIGH3 protein and macrophages in retinal endothelial cell apoptosis.Anti-vascular endothelial growth factor acts on retinal microglia/macrophage activation in a rat model of ocular inflammation Role of microglia adenosine A(2A) receptors in retinal and brain neurodegenerative diseases.The role of microglia in diabetic retinopathy Caveolin-1 increases proinflammatory chemoattractants and blood-retinal barrier breakdown but decreases leukocyte recruitment in inflammation.Inflammation in the pathogenesis of microvascular complications in diabetes.PKCζ mediates breakdown of outer blood-retinal barriers in diabetic retinopathy 7-Ketocholesterol increases retinal microglial migration, activation, and angiogenicity: a potential pathogenic mechanism underlying age-related macular degeneration The causes of hyperreflective dots in optical coherence tomography excluding diabetic macular edema and retinal venous occlusion§.BTBR ob/ob mice as a novel diabetic neuropathy model: Neurological characterization and gene expression analyses Natural history of age-related retinal lesions that precede AMD in mice fed high or low glycemic index diets.Loss of caveolin-1 impairs retinal function due to disturbance of subretinal microenvironment Diabetic macular oedema: pathophysiology, management challenges and treatment resistance.Neuroretinal Apoptosis as a Vascular Dysfunction in Diabetic Patients Loss of DJ-1 elicits retinal abnormalities, visual dysfunction, and increased oxidative stress in mice.Lymphocytic Microparticles Modulate Angiogenic Properties of Macrophages in Laser-induced Choroidal Neovascularization.A2E accumulation influences retinal microglial activation and complement regulation Reduced innervation and delayed re-innervation after epithelial wounding in type 2 diabetic Goto-Kakizaki rats Beta-adrenergic receptor agonist decreases VEGF levels through altered eNOS and PKC signaling in diabetic retina.Effects on high cholesterol-fed to liver, retina, hippocampus, and Harderian gland in Goto-Kakizaki rat.An Aqueous Extract of Radix Astragali, Angelica sinensis, and Panax notoginseng Is Effective in Preventing Diabetic Retinopathy.The retinal pigment epithelium as a gateway for monocyte trafficking into the eye.Multimodal Highlighting of Structural Abnormalities in Diabetic Rat and Human Corneas The significance of the increased expression of phosphorylated MeCP2 in the membranes from patients with proliferative diabetic retinopathy.Adenosine Deaminase-2-Induced Hyperpermeability in Human Retinal Vascular Endothelial Cells Is Suppressed by MicroRNA-146b-3p Viewing the choroid: where we stand, challenges and contradictions in diabetic retinopathy and diabetic macular oedema.Uveitic macular edema.A cross talk between class A scavenger receptor and receptor for advanced glycation end-products contributes to diabetic retinopathy.Caveolins and caveolae in ocular physiology and pathophysiology.IGFBP-3 reduces eNOS and PKCzeta phosphorylation, leading to lowered VEGF levels.ROCK-1 mediates diabetes-induced retinal pigment epithelial and endothelial cell blebbing: Contribution to diabetic retinopathy.Hyperreflective intraretinal spots in diabetics without and with nonproliferative diabetic retinopathy: an in vivo study using spectral domain OCT.Effects of sub-Tenon's capsule triamcinolone acetonide injection combined with microaneurysm photocoagulation on diabetic macular edema Foxp3+ Tregs are recruited to the retina to repair pathological angiogenesis.Pharmacological Tools to Activate Microglia and their Possible use to Study Neural Network Patho-physiology.Quercetin Mitigates Inflammatory Responses Induced by Vascular Endothelial Growth Factor in Mouse Retinal Photoreceptor Cells through Suppression of Nuclear Factor Kappa B.

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Microglia/macrophages migrate through retinal epithelium barrier by a transcellular route in diabetic retinopathy: role of PKCζ in the Goto Kakizaki rat model.

http://www.wikidata.org/entity/Q35168171

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The American Journal of Pathology

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Boubaker Omri

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Florian Sennlaub

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Inflammation, stress, and diabetes

ICAM-1 regulates neutrophil adhesion and transcellular migration of TNF-alpha-activated vascular endothelium under flow

Interaction between NO and COX pathways in retinal cells exposed to elevated glucose and retina of diabetic rats

Ceramide recruits and activates protein kinase C zeta (PKC zeta) within structured membrane microdomains

The chemokine system in diverse forms of macrophage activation and polarization

Microglia in the mouse retina alter the structure and function of retinal pigmented epithelial cells: a potential cellular interaction relevant to AMD.

Diabetic retinopathy and diabetic macular edema: pathophysiology, screening, and novel therapies.

Microglial changes occur without neural cell death in diabetic retinopathy.

CX3CR1-dependent subretinal microglia cell accumulation is associated with cardinal features of age-related macular degeneration.

Age-dependent accumulation of lipofuscin in perivascular and subretinal microglia in experimental mice.

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942-953

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