Neutrophil elastase is produced by pulmonary artery smooth muscle cells and is linked to neointimal lesions
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Molecular pathogenesis of pulmonary arterial hypertensionChallenges and Current Efforts in the Development of Biomarkers for Chronic Inflammatory and Remodeling Conditions of the LungsMicrovessel mechanobiology in pulmonary arterial hypertension: cause and effectPotential contribution of phenotypically modulated smooth muscle cells and related inflammation in the development of experimental obstructive pulmonary vasculopathy in ratsInflammation and immunity in the pathogenesis of pulmonary arterial hypertensionPulmonary artery smooth muscle cell endothelin-1 expression modulates the pulmonary vascular response to chronic hypoxiaBMPR2 preserves mitochondrial function and DNA during reoxygenation to promote endothelial cell survival and reverse pulmonary hypertensionCompartment-specific expression of collagens and their processing enzymes in intrapulmonary arteries of IPAH patientsThe Effects of Antigen-Specific IgG1 Antibody for the Pulmonary-Hypertension-Phenotype and B Cells for Inflammation in Mice Exposed to Antigen and Fine Particles from Air Pollution.Elafin Reverses Pulmonary Hypertension via Caveolin-1-Dependent Bone Morphogenetic Protein Signaling.ELANE mutations in cyclic and severe congenital neutropenia: genetics and pathophysiologyTau pathology-dependent remodelling of cerebral arteries precedes Alzheimer's disease-related microvascular cerebral amyloid angiopathyModeling and rescue of the vascular phenotype of Williams-Beuren syndrome in patient induced pluripotent stem cells.Novel Targets of Drug Treatment for Pulmonary Hypertension.Molecular pathogenesis and current pathology of pulmonary hypertension.Exploratory analysis of the neutrophil to lymphocyte ratio in patients with pulmonary arterial hypertensionPro-proliferative and inflammatory signaling converge on FoxO1 transcription factor in pulmonary hypertension.Murine Gammaherpesvirus 68: A Small Animal Model for Gammaherpesvirus-Associated Diseases.Codependence of Bone Morphogenetic Protein Receptor 2 and Transforming Growth Factor-β in Elastic Fiber Assembly and Its Perturbation in Pulmonary Arterial Hypertension.Infiltrating Myeloid Cells Exert Protumorigenic Actions via Neutrophil Elastase.Neutrophil elastase-induced elastin degradation mediates macrophage influx and lung injury in 60% O2-exposed neonatal rats.[Towards new targets for the treatment of pulmonary arterial hypertension : Importance of cell-cell communications].Elastin, arterial mechanics, and cardiovascular diseaseThe Role of Neutrophils and Neutrophil Elastase in Pulmonary Arterial Hypertension
P2860
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P2860
Neutrophil elastase is produced by pulmonary artery smooth muscle cells and is linked to neointimal lesions
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Neutrophil elastase is produce ...... s linked to neointimal lesions
@ast
Neutrophil elastase is produce ...... s linked to neointimal lesions
@en
type
label
Neutrophil elastase is produce ...... s linked to neointimal lesions
@ast
Neutrophil elastase is produce ...... s linked to neointimal lesions
@en
prefLabel
Neutrophil elastase is produce ...... s linked to neointimal lesions
@ast
Neutrophil elastase is produce ...... s linked to neointimal lesions
@en
P2093
P2860
P1476
Neutrophil elastase is produce ...... s linked to neointimal lesions
@en
P2093
Andrew Orton
Cristina M Alvira
Edda Spiekerkoetter
Gabriela Rodriguez-Colon
Hirofumi Sawada
Leila Haghighat
Lingli Wang
Marlene Rabinovitch
Mingming Zhao
Swati Acharya
P2860
P304
P356
10.1016/J.AJPATH.2011.05.051
P407
P577
2011-07-19T00:00:00Z