The reduction of R1, a novel repressor protein for monoamine oxidase A, in major depressive disorder - Wikidata - awgldk - TriplyDB

The reduction of R1, a novel repressor protein for monoamine oxidase A, in major depressive disorder

The reduction of R1, a novel repressor protein for monoamine oxidase A, in major depressive disorder

http://www.wikidata.org/entity/Q35171311

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Astrocyte pathology in major depressive disorder: insights from human postmortem brain tissue The 5-HT deficiency theory of depression: perspectives from a naturalistic 5-HT deficiency model, the tryptophan hydroxylase 2Arg439His knockin mouse The development, past achievements, and future directions of brain PET Dynamics of the ternary complex formed by c-Myc interactor JPO2, transcriptional co-activator LEDGF/p75, and chromatin.Evidence revealing deregulation of the KLF11-MAO A pathway in association with chronic stress and depressive disorders Relationship of monoamine oxidase-A distribution volume to postpartum depression and postpartum crying Mechanistic role for a novel glucocorticoid-KLF11 (TIEG2) protein pathway in stress-induced monoamine oxidase A expression Chronic Social Stress and Ethanol Increase Expression of KLF11, a Cell Death Mediator, in Rat Brain Monoamine Oxidase-A Occupancy by Moclobemide and Phenelzine: Implications for the Development of Monoamine Oxidase Inhibitors Evaluation of the Isoflavone Genistein as Reversible Human Monoamine Oxidase-A and -B Inhibitor Elevated Monoamine Oxidase-A Distribution Volume in Borderline Personality Disorder Is Associated With Severity Across Mood Symptoms, Suicidality, and Cognition Greater monoamine oxidase a binding in alcohol dependence.Greater monoamine oxidase a binding in perimenopausal age as measured with carbon 11-labeled harmine positron emission tomography.Elevated monoamine oxidase a binding during major depressive episodes is associated with greater severity and reversed neurovegetative symptoms.The expression of KLF11 (TIEG2), a monoamine oxidase B transcriptional activator in the prefrontal cortex of human alcohol dependence.Neuroimaging markers of cellular function in major depressive disorder: implications for therapeutics, personalized medicine, and prevention.A neuroscientific update on monoamine oxidase and its inhibitors.Modulation of monoamine oxidase (MAO) expression in neuropsychiatric disorders: genetic and environmental factors involved in type A MAO expression.Binge ethanol exposure increases the Krüppel-like factor 11-monoamine oxidase (MAO) pathway in rats: Examining the use of MAO inhibitors to prevent ethanol-induced brain injury Selective dietary supplementation in early postpartum is associated with high resilience against depressed mood.Systemic lipopolysaccharide-mediated alteration of cortical neuromodulation involves increases in monoamine oxidase-A and acetylcholinesterase activity The IFNγ-PKR pathway in the prefrontal cortex reactions to chronic excessive alcohol use.Diabetes-causing gene, kruppel-like factor 11, modulates the antinociceptive response of chronic ethanol intake.Kaempferia parviflora rhizome extract and Myristica fragrans volatile oil increase the levels of monoamine neurotransmitters and impact the proteomic profiles in the rat hippocampus: Mechanistic insights into their neuroprotective effects.Regulatory Pathways of Monoamine Oxidase A during Social Stress.Type A and B monoamine oxidases distinctly modulate signal transduction pathway and gene expression to regulate brain function and survival of neurons.Associations of depression, anxiety and antidepressants with histological severity of nonalcoholic fatty liver disease.No effect of oral tyrosine on total tyrosine levels in breast milk: implications for dietary supplementation in early postpartum.Synthesis of New Hydrazone Derivatives for MAO Enzymes Inhibitory Activity.Potential Roles of microRNAs in the Regulation of Monoamine Oxidase A in the Brain

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The reduction of R1, a novel repressor protein for monoamine oxidase A, in major depressive disorder

http://www.wikidata.org/entity/Q35171311

description

2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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The reduction of R1, a novel r ...... , in major depressive disorder

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Chandra Johnson

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Craig A Stockmeier

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Donald B Sittman

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George Jurjus

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Grazyna Rajkowska

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James C Overholser

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Jeffrey H Meyer

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Junming Wang

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Lesa Dieter

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Mark C Austin

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P2860

Identification of a novel c-Myc protein interactor, JPO2, with transforming activity in medulloblastoma cells

Glucocorticoid and androgen activation of monoamine oxidase A is regulated differently by R1 and Sp1

Aggressive behavior and altered amounts of brain serotonin and norepinephrine in mice lacking MAOA

R1, a novel repressor of the human monoamine oxidase A

Monoamine oxidase A and repressor R1 are involved in apoptotic signaling pathway

MAO-A and -B gene knock-out mice exhibit distinctly different behavior

High activity-related allele of MAO-A gene associated with depressed suicide in males

Pharmacological profile of antidepressants and related compounds at human monoamine transporters

Elevated monoamine oxidase a levels in the brain: an explanation for the monoamine imbalance of major depression

MAO-A gene polymorphisms are associated with major depression and sleep disturbance in males

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2139-2148

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10.1038/NPP.2011.105

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