Podocyte injury-driven intracapillary plasminogen activator inhibitor type 1 accelerates podocyte loss via uPAR-mediated β1-integrin endocytosis.

Podocyte injury-driven intracapillary plasminogen activator inhibitor type 1 accelerates podocyte loss via uPAR-mediated β1-integrin endocytosis.

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http://www.wikidata.org/entity/Q35178613
Podocyte endocytosis in the regulation of the glomerular filtration barrierThe FERM protein EPB41L5 regulates actomyosin contractility and focal adhesion formation to maintain the kidney filtration barrier.Novel Plasminogen Activator Inhibitor-1 Inhibitors Prevent Diabetic Kidney Injury in a Mouse Model.Podocyturia: A Clue for the Rational Use of Amiloride in Alport Renal Disease.Physiology and pathophysiology of the plasminogen system in the kidney.Focal segmental glomerulosclerosis; why does it occur segmentally?A novel plasminogen activator inhibitor-1 inhibitor, TM5441, protects against high-fat diet-induced obesity and adipocyte injury in mice.A tripartite complex of suPAR, APOL1 risk variants and αvβ3 integrin on podocytes mediates chronic kidney disease.In IgA Nephropathy, Glomerulosclerosis Is Associated with Increased Urinary CD80 Excretion and Urokinase-Type Plasminogen Activator Receptor-Positive Podocyturia.Local CD34-positive capillaries decrease in mouse models of kidney disease associating with the severity of glomerular and tubulointerstitial lesions.In Acute IgA Nephropathy, Proteinuria and Creatinine Are in the Spot, but Podocyturia Operates in Silence: Any Place for Amiloride?Complex glomerular pathology of thrombotic microangiopathy and focal segmental glomerulosclerosis forms tumor-like mass in a renal transplant donor with severe renovascular hypertension.Serum suPAR and syndecan-4 levels predict severity of community-acquired pneumonia: a prospective, multi-centre study.Modified scanning electron microscopy reveals pathological crosstalk between endothelial cells and podocytes in a murine model of membranoproliferative glomerulonephritis.Focal segmental glomerulosclerosis lesion associated with inhibition of tyrosine kinases by lenvatinib: a case reportErzhi Formula Extracts Reverse Renal Injury in Diabetic Nephropathy Rats by Protecting the Renal Podocytes
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Podocyte injury-driven intracapillary plasminogen activator inhibitor type 1 accelerates podocyte loss via uPAR-mediated β1-integrin endocytosis.

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http://www.wikidata.org/entity/Q35178613
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2015 nî lūn-bûn
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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած
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2015 թվականի հունվարին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@ast
Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@en
type
label
Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@ast
Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@en
prefLabel
Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@ast
Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@en
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Podocyte injury-driven intraca ...... iated β1-integrin endocytosis.
@en
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10.1152/AJPRENAL.00616.2014
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