Chronic NOS inhibition accelerates NAFLD progression in an obese rat model. - Wikidata - awgldk - TriplyDB

Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

http://www.wikidata.org/entity/Q35178651

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Deficiency of eNOS exacerbates early-stage NAFLD pathogenesis by changing the fat distribution Identification of cysteinylated transthyretin, a predictive biomarker of treatment response to partially hydrolyzed guar gum in type 2 diabetes rats, by surface-enhanced laser desorption/ionization time-of-flight mass spectrometry.Ablation of eNOS does not promote adipose tissue inflammation.Clinical implications of understanding the association between oxidative stress and pediatric NAFLD.ER Stress Inhibits Liver Fatty Acid Oxidation while Unmitigated Stress Leads to Anorexia-Induced Lipolysis and Both Liver and Kidney Steatosis.Early Effects of a Low Fat, Fructose-Rich Diet on Liver Metabolism, Insulin Signaling, and Oxidative Stress in Young and Adult Rats.Oral nitrite restores age-dependent phenotypes in eNOS-null mice

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P2860

Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

http://www.wikidata.org/entity/Q35178651

description

2015 nî lūn-bûn

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2015 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հունվարին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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type

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Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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prefLabel

Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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AJPGI.00247.2014

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American Journal of Physiology - Gastrointestinal and Liver Physiology

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Chronic NOS inhibition accelerates NAFLD progression in an obese rat model.

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P2093

Jaume Padilla

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M Harold Laughlin

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Nathan T Jenkins

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R Scott Rector

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P2860

High protonic potential actuates a mechanism of production of reactive oxygen species in mitochondria

Regulation of mitochondrial respiration by nitric oxide inhibition of cytochrome c oxidase.

Reduced hepatic eNOS phosphorylation is associated with NAFLD and type 2 diabetes progression and is prevented by daily exercise in hyperphagic OLETF rats

Akt-dependent phosphorylation of endothelial nitric-oxide synthase mediates penile erection

Mitochondrial dysfunction precedes insulin resistance and hepatic steatosis and contributes to the natural history of non-alcoholic fatty liver disease in an obese rodent model.

Nitric oxide inhibition of respiration involves both competitive (heme) and noncompetitive (copper) binding to cytochrome c oxidase

Nitric oxide donors and cardiovascular agents modulating the bioactivity of nitric oxide: an overview.

Daily exercise vs. caloric restriction for prevention of nonalcoholic fatty liver disease in the OLETF rat model.

Endothelial NO/cGMP/VASP signaling attenuates Kupffer cell activation and hepatic insulin resistance induced by high-fat feeding

Effects of chronic nitric oxide synthase inhibition on responses to acute exercise in swine

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