Gastric bypass surgery enhances glucagon-like peptide 1-stimulated postprandial insulin secretion in humans - Wikidata - awgldk - TriplyDB

Gastric bypass surgery enhances glucagon-like peptide 1-stimulated postprandial insulin secretion in humans

Gastric bypass surgery enhances glucagon-like peptide 1-stimulated postprandial insulin secretion in humans

http://www.wikidata.org/entity/Q35179981

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Regulation of glucose homeostasis by GLP-1 The Use of Rat and Mouse Models in Bariatric Surgery Experiments Is GLP-1 a hormone: Whether and When?Effects of glucagon like peptide-1 to mediate glycemic effects of weight loss surgery Can medical therapy mimic the clinical efficacy or physiological effects of bariatric surgery?Do we really know why diabetes remits after gastric bypass surgery?Physiology of proglucagon peptides: role of glucagon and GLP-1 in health and disease Remodeling of the residual gastric mucosa after roux-en-y gastric bypass or vertical sleeve gastrectomy in diet-induced obese rats Glucagon-Like Peptide-1 and Its Class B G Protein-Coupled Receptors: A Long March to Therapeutic Successes A gut feeling to cure diabetes: potential mechanisms of diabetes remission after bariatric surgery The role of gut adaptation in the potent effects of multiple bariatric surgeries on obesity and diabetes Gut-Brain Cross-Talk in Metabolic Control.Altered islet function and insulin clearance cause hyperinsulinemia in gastric bypass patients with symptoms of postprandial hypoglycemia The sum of many parts: potential mechanisms for improvement in glucose homeostasis after bariatric surgery Evidence from a single individual that increased plasma GLP-1 and GLP-1-stimulated insulin secretion after gastric bypass are independent of foregut exclusion Surgical treatment of nonalcoholic fatty liver disease in severely obese patients Metabolic bariatric surgery and type 2 diabetes mellitus: an endocrinologist's perspective.Weight Loss and the Prevention and Treatment of Type 2 Diabetes Using Lifestyle Therapy, Pharmacotherapy, and Bariatric Surgery: Mechanisms of Action.Postprandial insulin secretion after gastric bypass surgery: the role of glucagon-like peptide 1.Insulin response to oral stimuli and glucose effectiveness increased in neuroglycopenia following gastric bypass GLP-1 plays a limited role in improved glycemia shortly after Roux-en-Y gastric bypass: a comparison with intensive lifestyle modification Preserved Insulin Secretory Capacity and Weight Loss Are the Predominant Predictors of Glycemic Control in Patients With Type 2 Diabetes Randomized to Roux-en-Y Gastric Bypass Improved Insulin Sensitivity 3 Months After RYGB Surgery Is Associated With Increased Subcutaneous Adipose Tissue AMPK Activity and Decreased Oxidative Stress.Comparison Review of Short-Acting and Long-Acting Glucagon-like Peptide-1 Receptor Agonists Gastric bypass alters both glucose-dependent and glucose-independent regulation of islet hormone secretion.GLP-1 receptor antagonist exendin-(9-39) elevates fasting blood glucose levels in congenital hyperinsulinism owing to inactivating mutations in the ATP-sensitive K+ channel Effect of glycemia on plasma incretins and the incretin effect during oral glucose tolerance test.Alterations in gastrointestinal, endocrine, and metabolic processes after bariatric Roux-en-Y gastric bypass surgery Suppression of food intake by glucagon-like peptide-1 receptor agonists: relative potencies and role of dipeptidyl peptidase-4 Gastric bypass and banding equally improve insulin sensitivity and β cell function Comparison of Bariatric Surgical Procedures for Diabetes Remission: Efficacy and Mechanisms Postprandial Hypoglycemia in Children after Gastric Surgery: Clinical Characterization and Pathophysiology.Insulinoma After Bariatric Surgery: Diagnostic Dilemma and Therapeutic Approaches.GLP-1 action and glucose tolerance in subjects with remission of type 2 diabetes after gastric bypass surgery Peripheral, but not central, GLP-1 receptor signaling is required for improvement in glucose tolerance after Roux-en-Y gastric bypass in mice Effects of gastrogastric fistula repair on weight loss and gut hormone levels Vertical sleeve gastrectomy is effective in two genetic mouse models of glucagon-like Peptide 1 receptor deficiency.Incretin therapy and islet pathology: a time for caution.Metabolic effects of bariatric surgery in patients with moderate obesity and type 2 diabetes: analysis of a randomized control trial comparing surgery with intensive medical treatment Inactivation of specific β cell transcription factors in type 2 diabetes.

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Gastric bypass surgery enhances glucagon-like peptide 1-stimulated postprandial insulin secretion in humans

http://www.wikidata.org/entity/Q35179981

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2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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David A D'Alessio

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Marzieh Salehi

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Ronald L Prigeon

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P2860

NIH conference. Gastrointestinal surgery for severe obesity. Consensus Development Conference Panel

Acute effects of gastric bypass versus gastric restrictive surgery on beta-cell function and insulinotropic hormones in severely obese patients with type 2 diabetes.

Effect of endogenous GLP-1 on insulin secretion in type 2 diabetes.

Isolation and characterization of exendin-4, an exendin-3 analogue, from Heloderma suspectum venom. Further evidence for an exendin receptor on dispersed acini from guinea pig pancreas.

Prevalence of overweight and obesity among US children, adolescents, and adults, 1999-2002.

Insulin within islets is a physiologic glucagon release inhibitor

Evaluation and management of adult hypoglycemic disorders: an Endocrine Society Clinical Practice Guideline.

Gut hormones and diabetes mellitus.

Surgery for severe obesity.

beta-cell function in obesity: effects of weight loss.

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10.2337/DB11-0203

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