Signaling between SR and plasmalemma in smooth muscle: sparks and the activation of Ca2+-sensitive ion channels.
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Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle DisordersMolecular mechanisms of renal blood flow autoregulationBK channels: multiple sensors, one activation gateIon channel remodeling in vascular smooth muscle during hypertension: Implications for novel therapeutic approachesQuercetin relaxes rat tail main artery partly via a PKG-mediated stimulation of KCa 1.1 channelsThe BK channel accessory beta1 subunit determines alcohol-induced cerebrovascular constrictionPharmacological inhibition of TRPM4 hyperpolarizes vascular smooth muscleSub-plasmalemmal [Ca2+]i upstroke in myocytes of the guinea-pig small intestine evoked by muscarinic stimulation: IP3R-mediated Ca2+ release induced by voltage-gated Ca2+ entryReduced Ca2+ spark activity after subarachnoid hemorrhage disables BK channel control of cerebral artery tone.Contribution of electromechanical coupling between Kv and Ca v1.2 channels to coronary dysfunction in obesity.Traumatic brain injury disrupts cerebrovascular tone through endothelial inducible nitric oxide synthase expression and nitric oxide gain of functionUsing total fluorescence increase (signal mass) to determine the Ca2+ current underlying localized Ca2+ events.RACK1 is a BKCa channel binding protein.Role of BK(Ca) channels in cephalic vasodilation induced by CGRP, NO and transcranial electrical stimulation in the rat.Transient receptor potential melastatin 8 channel involvement in the regulation of vascular tone.Propofol increases the Ca2+ sensitivity of BKCa in the cerebral arterial smooth muscle cells of mice.Impact of subarachnoid hemorrhage on local and global calcium signaling in cerebral artery myocytes.Cell surface topology creates high Ca2+ signalling microdomains.Differential regulation and recovery of intracellular Ca2+ in cerebral and small mesenteric arterial smooth muscle cells of simulated microgravity rat.Subtype identification and functional characterization of ryanodine receptors in rat cerebral artery myocytes.Interactions between calcium and reactive oxygen species in pulmonary arterial smooth muscle responses to hypoxia.Pituitary adenylate cyclase activating polypeptide (PACAP) dilates cerebellar arteries through activation of large-conductance Ca(2+)-activated (BK) and ATP-sensitive (K ATP) K (+) channels.Ion channels in smooth muscle: regulators of intracellular calcium and contractility.BK channels regulate sinoatrial node firing rate and cardiac pacing in vivoPotassium channels in the peripheral microcirculation.Hypoxia. 4. Hypoxia and ion channel functionTuning magnesium sensitivity of BK channels by mutations.Detection of differentially regulated subsarcolemmal calcium signals activated by vasoactive agonists in rat pulmonary artery smooth muscle cells.Regulation of gastrointestinal motility by Ca2+/calmodulin-stimulated protein kinase II.Type 2 ryanodine receptors are highly sensitive to alcoholCalcium signaling in smooth muscle.Molecular and functional significance of Ca(2+)-activated Cl(-) channels in pulmonary arterial smooth muscle.Endogenous cytosolic Ca(2+) buffering is necessary for TRPM4 activity in cerebral artery smooth muscle cells.Electrical slow waves in the mouse oviduct are dependent upon a calcium activated chloride conductance encoded by Tmem16a.Tacrolimus inhibits vasoconstriction by increasing Ca(2+) sparks in rat aorta.Reconstruction of Cell Surface Densities of Ion Pumps, Exchangers, and Channels from mRNA Expression, Conductance Kinetics, Whole-Cell Calcium, and Current-Clamp Voltage Recordings, with an Application to Human Uterine Smooth Muscle Cells.Signal-transduction pathways that regulate smooth muscle function. II. Receptor-ion channel coupling mechanisms in gastrointestinal smooth muscle.Impaired propulsive motility in the distal but not proximal colon of BK channel β1-subunit knockout mice.Targeting Ion Channels: An Important Therapeutic Implication in Gastrointestinal Dysmotility in Patients With Spinal Cord InjuryCalcium events in smooth muscles and their interstitial cells; physiological roles of sparks.
P2860
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P2860
Signaling between SR and plasmalemma in smooth muscle: sparks and the activation of Ca2+-sensitive ion channels.
description
2003 nî lūn-bûn
@nan
2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@ast
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@en
type
label
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@ast
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@en
prefLabel
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@ast
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@en
P1433
P1476
Signaling between SR and plasm ...... f Ca2+-sensitive ion channels.
@en
P2093
George C Wellman
Mark T Nelson
P304
P356
10.1016/S0143-4160(03)00124-6
P577
2003-09-01T00:00:00Z