Loss of cation-chloride cotransporter expression in preterm infants with white matter lesions: implications for the pathogenesis of epilepsy
about
Age- and sex-dependent susceptibility to phenobarbital-resistant neonatal seizures: role of chloride co-transportersAcute TrkB inhibition rescues phenobarbital-resistant seizures in a mouse model of neonatal ischemiaNeuronal chloride accumulation and excitatory GABA underlie aggravation of neonatal epileptiform activities by phenobarbital.Complex pattern of interaction between in utero hypoxia-ischemia and intra-amniotic inflammation disrupts brain development and motor function.GABA, resting-state connectivity and the developing brain.Erythropoietin attenuates loss of potassium chloride co-transporters following prenatal brain injury.Cation-chloride cotransporters in neuronal development, plasticity and diseaseLate development of the GABAergic system in the human cerebral cortex and white matterPostnatal Erythropoietin Mitigates Impaired Cerebral Cortical Development Following Subplate Loss from Prenatal Hypoxia-Ischemia.Prenatal Hypoxia-Ischemia Induces Abnormalities in CA3 Microstructure, Potassium Chloride Co-Transporter 2 Expression and Inhibitory Tone.Erythropoietin Modulates Cerebral and Serum Degradation Products from Excess Calpain Activation following Prenatal Hypoxia-Ischemia.Impaired neuronal KCC2 function by biallelic SLC12A5 mutations in migrating focal seizures and severe developmental delayImaging and serum biomarkers reflecting the functional efficacy of extended erythropoietin treatment in rats following infantile traumatic brain injury.Neurogenesis and maturation in neonatal brain injury.Preclinical Models of Encephalopathy of Prematurity.Developmental Expression Patterns of KCC2 and Functionally Associated Molecules in the Human Brain.Chloride cotransporter NKCC1 inhibitor bumetanide protects against white matter injury in a rodent model of periventricular leukomalacia.The KCC2 Cotransporter and Human Epilepsy: Getting Excited About Inhibition.Hemodynamic and Metabolic Assessment of Neonates With Punctate White Matter Lesions Using Phase-Contrast MRI and T2-Relaxation-Under-Spin-Tagging (TRUST) MRI.Mild Intrauterine Hypoperfusion Leads to Lumbar and Cortical Hyperexcitability, Spasticity, and Muscle Dysfunctions in Rats: Implications for Prematurity.
P2860
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P2860
Loss of cation-chloride cotransporter expression in preterm infants with white matter lesions: implications for the pathogenesis of epilepsy
description
2010 nî lūn-bûn
@nan
2010 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@ast
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@en
type
label
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@ast
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@en
prefLabel
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@ast
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@en
P2093
P2860
P1476
Loss of cation-chloride cotran ...... r the pathogenesis of epilepsy
@en
P2093
Ian Thompson
Irina Mikolaenko
Mark L Cohen
Monisha Goyal
Shenandoah Robinson
P2860
P304
P356
10.1097/NEN.0B013E3181DD25BC
P577
2010-06-01T00:00:00Z