Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK. - Wikidata - awgldk - TriplyDB

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

http://www.wikidata.org/entity/Q35190872

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Effect of methotrexate on bone and wound healing.Methotrexate inhibits NF-κB activity via long intergenic (noncoding) RNA-p21 induction The microbial capsular polysaccharide galactoxylomannan inhibits IL-17A production in circulating T cells from rheumatoid arthritis patients.Methotrexate-mediated inhibition of nuclear factor κB activation by distinct pathways in T cells and fibroblast-like synoviocytes.Defective structural RNA processing in relapsing-remitting multiple sclerosis Methotrexate increases expression of cell cycle checkpoint genes via JNK activation Genome-scale case-control analysis of CD4+ T-cell DNA methylation in juvenile idiopathic arthritis reveals potential targets involved in disease.Systemic Wound Healing Associated with local sub-Cutaneous Mechanical Stimulation.Teriflunomide and its mechanism of action in multiple sclerosis.Current understanding of rheumatoid arthritis therapy.Methotrexate induces production of IL-1 and IL-6 in the monocytic cell line U937.Mechanism of action of methotrexate in rheumatoid arthritis, and the search for biomarkers.LD-aminopterin in the canine homologue of human atopic dermatitis: a randomized, controlled trial reveals dosing factors affecting optimal therapy.Multi-omic landscape of rheumatoid arthritis: re-evaluation of drug adverse effects.Methotrexate analogues display enhanced inhibition of TNF-α production in whole blood from RA patients.Impact of methotrexate on oxidative stress and apoptosis markers in psoriatic patients.Increased baseline RUNX2, caspase 3 and p21 gene expressions in the peripheral blood of disease-modifying anti-rheumatic drug-naïve rheumatoid arthritis patients are associated with improved clinical response to methotrexate therapy.Alpha2beta1 Integrin (VLA-2) Protects Activated Human Effector T Cells From Methotrexate-Induced Apoptosis

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P2860

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

http://www.wikidata.org/entity/Q35190872

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

@ast

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

@en

type

label

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

@ast

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

@en

prefLabel

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

@ast

Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.

@en

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P1433

Arthritis and Rheumatism

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Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK

@en

P2093

Charles F Spurlock

http://www.w3.org/2001/XMLSchema#string

Jessica P Aune

http://www.w3.org/2001/XMLSchema#string

John T Tossberg

http://www.w3.org/2001/XMLSchema#string

Joseph W Huston

http://www.w3.org/2001/XMLSchema#string

Nancy J Olsen

http://www.w3.org/2001/XMLSchema#string

Patrick L Collins

http://www.w3.org/2001/XMLSchema#string

Thomas M Aune

http://www.w3.org/2001/XMLSchema#string

Zachary T Aune

http://www.w3.org/2001/XMLSchema#string

P2860

The roles of ASK family proteins in stress responses and diseases

Selective interaction of JNK protein kinase isoforms with transcription factors

Critical role for tetrahydrobiopterin recycling by dihydrofolate reductase in regulation of endothelial nitric-oxide synthase coupling: relative importance of the de novo biopterin synthesis versus salvage pathways

Molecular action of methotrexate in inflammatory diseases

Adenosine receptors as therapeutic targets

JNK1 is not essential for TNF-mediated joint disease.

The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

Death receptors: signaling and modulation

A2A adenosine receptor and its modulators: overview on a druggable GPCR and on structure-activity relationship analysis and binding requirements of agonists and antagonists

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2606-2616

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scholarly article

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10.1002/ART.30457

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9

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63

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2011-09-01T00:00:00Z

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21618198

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3165146

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