IglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVS - Wikidata - awgldk - TriplyDB

IglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVS

IglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVS

http://www.wikidata.org/entity/Q35191992

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From the Outside-In: The Francisella tularensis Envelope and Virulence Francisella tularensis IglG Belongs to a Novel Family of PAAR-Like T6SS Proteins and Harbors a Unique N-terminal Extension Required for Virulence Importance of PdpC, IglC, IglI, and IglG for modulation of a host cell death pathway induced by Francisella tularensis Structure-Function Analysis of DipA, a Francisella tularensis Virulence Factor Required for Intracellular Replication FipB, an essential virulence factor of Francisella tularensis subsp. tularensis, has dual roles in disulfide bond formation.Production of outer membrane vesicles and outer membrane tubes by Francisella novicida.DotU and VgrG, core components of type VI secretion systems, are essential for Francisella LVS pathogenicity Identification of mechanisms for attenuation of the FSC043 mutant of Francisella tularensis SCHU S4.Bacterial programming of host responses: coordination between type I interferon and cell death.Unique substrates secreted by the type VI secretion system of Francisella tularensis during intramacrophage infection.The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infection Identification of Francisella novicida mutants that fail to induce prostaglandin E(2) synthesis by infected macrophages.A method for functional trans-complementation of intracellular Francisella tularensis.The biochemical properties of the Francisella pathogenicity island (FPI)-encoded proteins IglA, IglB, IglC, PdpB and DotU suggest roles in type VI secretion.Disruption of Francisella tularensis Schu S4 iglI, iglJ, and pdpC genes results in attenuation for growth in human macrophages and in vivo virulence in mice and reveals a unique phenotype for pdpC.Lack of OxyR and KatG Results in Extreme Susceptibility of Francisella tularensis LVS to Oxidative Stress and Marked Attenuation In vivo.Fighting outbreaks with bacterial genomics: case review and workflow proposal.Differential Growth of Francisella tularensis, Which Alters Expression of Virulence Factors, Dominant Antigens, and Surface-Carbohydrate Synthases, Governs the Apparent Virulence of Ft SchuS4 to Immunized Animals.Microinjection of Francisella tularensis and Listeria monocytogenes reveals the importance of bacterial and host factors for successful replication Francisella Inflammasomes: Integrated Responses to a Cytosolic Stealth Bacterium.A mutagenesis-based approach identifies amino acids in the N-terminal part of Francisella tularensis IglE that critically control Type VI system-mediated secretion.Francisella tularensis type VI secretion system comes of age.The Francisella Type VI Secretion System.

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P2860

IglG and IglI of the Francisella pathogenicity island are important virulence determinants of Francisella tularensis LVS

http://www.wikidata.org/entity/Q35191992

description

2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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IglG and IglI of the Francisel ...... of Francisella tularensis LVS

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IglG and IglI of the Francisel ...... of Francisella tularensis LVS

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IglG and IglI of the Francisel ...... of Francisella tularensis LVS

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IglG and IglI of the Francisel ...... of Francisella tularensis LVS

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IglG and IglI of the Francisel ...... of Francisella tularensis LVS

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Infection and Immunity

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IglG and IglI of the Francisel ...... of Francisella tularensis LVS

@en

P2093

Jeanette E Bröms

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Lena Meyer

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Moa Lavander

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P2860

MiR-155 induction by F. novicida but not the virulent F. tularensis results in SHIP down-regulation and enhanced pro-inflammatory cytokine response

Identification of a conserved bacterial protein secretion system in Vibrio cholerae using the Dictyostelium host model system

A virulence locus of Pseudomonas aeruginosa encodes a protein secretion apparatus

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Type VI secretion is a major virulence determinant in Burkholderia mallei

Type VI secretion system translocates a phage tail spike-like protein into target cells where it cross-links actin

Mutations of Francisella novicida that alter the mechanism of its phagocytosis by murine macrophages

TLR-dependent control of Francisella tularensis infection and host inflammatory responses

Adaptation of Francisella tularensis to the mammalian environment is governed by cues which can be mimicked in vitro.

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3683-3696

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scholarly article

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10.1128/IAI.01344-10

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9

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79

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Anders Sjöstedt

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2011-06-20T00:00:00Z

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21690239

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3165494

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