The NF-kappa B signal transduction pathway in aortic endothelial cells is primed for activation in regions predisposed to atherosclerotic lesion formation.
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A novel role of CCN3 in regulating endothelial inflammationCanonical pathway of nuclear factor kappa B activation selectively regulates proinflammatory and prothrombotic responses in human atherosclerosisEpigenetic associations in relation to cardiovascular prevention and therapeuticsMetastasis of circulating tumor cells: favorable soil or suitable biomechanics, or both?Chemokines and microRNAs in atherosclerosisEndothelial epigenetics in biomechanical stress: disturbed flow-mediated epigenomic plasticity in vivo and in vitroThe role of notch in the cardiovascular system: potential adverse effects of investigational notch inhibitorsRedox Roles of Reactive Oxygen Species in Cardiovascular DiseasesSite-specific microRNA-92a regulation of Kruppel-like factors 4 and 2 in atherosusceptible endotheliumNF-kappaB: pivotal mediator or innocent bystander in atherogenesis?Rapid activation of nuclear factor-kappaB in airway epithelium in a murine model of allergic airway inflammationThe integrin alpha2beta1 agonist, aggretin, promotes proliferation and migration of VSMC through NF-kB translocation and PDGF productionHIF-1α is required for disturbed flow-induced metabolic reprogramming in human and porcine vascular endotheliumRecent insights into the cellular biology of atherosclerosisFlow-dependent cellular mechanotransduction in atherosclerosis.Human haemodynamic frequency harmonics regulate the inflammatory phenotype of vascular endothelial cells.A leukocyte-mimetic magnetic resonance imaging contrast agent homes rapidly to activated endothelium and tracks with atherosclerotic lesion macrophage contentFocal adhesion kinase modulates activation of NF-kappaB by flow in endothelial cells.Endothelial cell PECAM-1 promotes atherosclerotic lesions in areas of disturbed flow in ApoE-deficient miceAtherosclerosis-prone hemodynamics differentially regulates endothelial and smooth muscle cell phenotypes and promotes pro-inflammatory priming.Control of tissue morphology by Fasciclin III-mediated intercellular adhesion.Quantitative 3D fluorescence technique for the analysis of en face preparations of arterial walls using quantum dot nanocrystals and two-photon excitation laser scanning microscopy.Hemodynamics in the mouse aortic arch as assessed by MRI, ultrasound, and numerical modeling.Increased endothelial expression of Toll-like receptor 2 at sites of disturbed blood flow exacerbates early atherogenic events.Stat3-dependent acute Rantes production in vascular smooth muscle cells modulates inflammation following arterial injury in mice.Nuclear factor-{kappa}B activation contributes to vascular endothelial dysfunction via oxidative stress in overweight/obese middle-aged and older humans.Chicoric acid is a potent anti-atherosclerotic ingredient by anti-oxidant action and anti-inflammation capacity.Systemic delivery of microRNA-181b inhibits nuclear factor-κB activation, vascular inflammation, and atherosclerosis in apolipoprotein E-deficient mice.Hemodynamic shear stress and the endothelium in cardiovascular pathophysiologyLanthanum chloride suppresses oxysterol-induced ECV-304 cell apoptosis via inhibition of intracellular Ca(2+) concentration elevation, oxidative stress, and activation of ERK and NF-κB signaling pathways.Insulin resistance, lipotoxicity, type 2 diabetes and atherosclerosis: the missing links. The Claude Bernard Lecture 2009Beneficial effects of pomegranate juice on oxidation-sensitive genes and endothelial nitric oxide synthase activity at sites of perturbed shear stress.Myeloid IκBα deficiency promotes atherogenesis by enhancing leukocyte recruitment to the plaquesElastomeric microposts integrated into microfluidics for flow-mediated endothelial mechanotransduction analysis.Quercetin is a potent anti-atherosclerotic compound by activation of SIRT1 signaling under oxLDL stimulation.Artery buckling stimulates cell proliferation and NF-κB signaling.MicroRNA-10a regulation of proinflammatory phenotype in athero-susceptible endothelium in vivo and in vitro.The antiinflammatory effect of laminar flow: the role of PPARgamma, epoxyeicosatrienoic acids, and soluble epoxide hydrolaseThe role of endoplasmic reticulum stress in the progression of atherosclerosis.Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives
P2860
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P2860
The NF-kappa B signal transduction pathway in aortic endothelial cells is primed for activation in regions predisposed to atherosclerotic lesion formation.
description
2000 nî lūn-bûn
@nan
2000 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
The NF-kappa B signal transduc ...... erosclerotic lesion formation.
@ast
The NF-kappa B signal transduc ...... erosclerotic lesion formation.
@en
type
label
The NF-kappa B signal transduc ...... erosclerotic lesion formation.
@ast
The NF-kappa B signal transduc ...... erosclerotic lesion formation.
@en
prefLabel
The NF-kappa B signal transduc ...... erosclerotic lesion formation.
@ast
The NF-kappa B signal transduc ...... erosclerotic lesion formation.
@en
P2093
P2860
P921
P356
P1476
The NF-kappa B signal transduc ...... herosclerotic lesion formation
@en
P2093
P2860
P304
P356
10.1073/PNAS.97.16.9052
P407
P577
2000-08-01T00:00:00Z