On the significance of the role of cellular stress response reactions in the toxic actions of dioxin.
about
Dioxin revisited: developments since the 1997 IARC classification of dioxin as a human carcinogenHarmaline and harmalol inhibit the carcinogen-activating enzyme CYP1A1 via transcriptional and posttranslational mechanismsGene-chemical interactions in the developing mammalian nervous system: Effects on proliferation, neurogenesis and differentiationDiabetes, metabolic syndrome, and obesity in relation to serum dioxin concentrations: the Seveso women's health studyAssociation of dioxin and other persistent organic pollutants (POPs) with diabetes: epidemiological evidence and new mechanisms of beta cell dysfunctionMolecular epidemiologic evidence for diabetogenic effects of dioxin exposure in U.S. Air force veterans of the Vietnam warMechanisms of exocrine pancreatic toxicity induced by oral treatment with 2,3,7,8-tetrachlorodibenzo-p-dioxin in female Harlan Sprague-Dawley RatsCharacterization of MCF mammary epithelial cells overexpressing the Arylhydrocarbon receptor (AhR).Characterization of the pattern of the nongenomic signaling pathway through which TCDD-induces early inflammatory responses in U937 human macrophagesQuercetin blocks caveolae-dependent pro-inflammatory responses induced by co-planar PCBs.Differential developmental toxicity of naphthoic acid isomers in medaka (Oryzias latipes) embryos.Gut feelings of safety: tolerance to the microbiota mediated by innate immune receptors.Molecular targets that link dioxin exposure to toxicity phenotypes.Dioxin-mediated tumor progression through activation of mitochondria-to-nucleus stress signaling.Evidence supporting the hypothesis that one of the main functions of the aryl hydrocarbon receptor is mediation of cell stress responses.Coplanar polychlorinated biphenyl-induced CYP1A1 is regulated through caveolae signaling in vascular endothelial cellsThe conversion of rapid TCCD nongenomic signals to persistent inflammatory effects via select protein kinases in MCF10A cellsA new cross-talk between the aryl hydrocarbon receptor and RelB, a member of the NF-kappaB familyTCDD-induced cyclooxygenase-2 expression is mediated by the nongenomic pathway in mouse MMDD1 macula densa cells and kidneys.Development of a human adipocyte model derived from human mesenchymal stem cells (hMSC) as a tool for toxicological studies on the action of TCDD.Non-genomic action of TCDD to induce inflammatory responses in HepG2 human hepatoma cells and in liver of C57BL/6J mice.Dioxin increases C/EBPbeta transcription by activating cAMP/protein kinase A.
P2860
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P2860
On the significance of the role of cellular stress response reactions in the toxic actions of dioxin.
description
2003 nî lūn-bûn
@nan
2003 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
On the significance of the rol ...... n the toxic actions of dioxin.
@ast
On the significance of the rol ...... n the toxic actions of dioxin.
@en
type
label
On the significance of the rol ...... n the toxic actions of dioxin.
@ast
On the significance of the rol ...... n the toxic actions of dioxin.
@en
prefLabel
On the significance of the rol ...... n the toxic actions of dioxin.
@ast
On the significance of the rol ...... n the toxic actions of dioxin.
@en
P1476
On the significance of the rol ...... n the toxic actions of dioxin.
@en
P2093
Fumio Matsumura
P304
P356
10.1016/S0006-2952(03)00157-6
P407
P577
2003-08-01T00:00:00Z