Sensitization of nociceptive spinal neurons contributes to pain in a transgenic model of sickle cell disease.
about
Cognitive and behavior deficits in sickle cell mice are associated with profound neuropathologic changes in hippocampus and cerebellum.Updated Mechanisms of Sickle Cell Disease-Associated Chronic painEndogenous BDNF augments NMDA receptor phosphorylation in the spinal cord via PLCγ, PKC, and PI3K/Akt pathways during colitisComparative Analysis of Pain Behaviours in Humanized Mouse Models of Sickle Cell Anemia.An Evaluation of Central Sensitization in Patients With Sickle Cell Disease.New insights into sickle cell disease: mechanisms and investigational therapies.Frequency of Hospitalizations for Pain and Association With Altered Brain Network Connectivity in Sickle Cell Disease.Cannabinoid receptor-specific mechanisms to alleviate pain in sickle cell anemia via inhibition of mast cell activation and neurogenic inflammation.Electroacupuncture in conscious free-moving mice reduces pain by ameliorating peripheral and central nociceptive mechanisms.Characterization of functional brain activity and connectivity using EEG and fMRI in patients with sickle cell disease.Chronic Opioid Therapy and Central Sensitization in Sickle Cell Disease.Small-molecule nociceptin receptor agonist ameliorates mast cell activation and pain in sickle mice.Spinal glial activation and oxidative stress are alleviated by treatment with curcumin or coenzyme Q in sickle mice.Targeting novel mechanisms of pain in sickle cell disease.CaMKIIα underlies spontaneous and evoked pain behaviors in Berkeley sickle cell transgenic mice.Individuals with sickle cell disease have a significantly greater vasoconstriction response to thermal pain than controls and have significant vasoconstriction in response to anticipation of pain.Transcriptomic analysis of gene signatures associated with sickle pain.Characterization of a mouse model of sickle cell trait: parallels to human trait and a novel finding of cutaneous sensitization.Mast cell-neural interactions contribute to pain and itch.Bivalent ligand MCC22 potently attenuates nociception in a murine model of sickle cell disease.Endothelin-Type A receptors mediate pain in a mouse model of sickle cell disease.Sensitization of C-fiber nociceptors in mice with sickle cell disease is decreased by local inhibition of anandamide hydrolysis.Sickle cell disease: a natural model of acute and chronic pain.
P2860
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P2860
Sensitization of nociceptive spinal neurons contributes to pain in a transgenic model of sickle cell disease.
description
2015 nî lūn-bûn
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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Sensitization of nociceptive s ...... model of sickle cell disease.
@ast
Sensitization of nociceptive s ...... model of sickle cell disease.
@en
type
label
Sensitization of nociceptive s ...... model of sickle cell disease.
@ast
Sensitization of nociceptive s ...... model of sickle cell disease.
@en
prefLabel
Sensitization of nociceptive s ...... model of sickle cell disease.
@ast
Sensitization of nociceptive s ...... model of sickle cell disease.
@en
P2093
P2860
P1433
P1476
Sensitization of nociceptive s ...... model of sickle cell disease.
@en
P2093
Donald A Simone
Giuseppe Cataldo
Kalpna Gupta
Sugandha Rajput
P2860
P304
P356
10.1097/J.PAIN.0000000000000104
P407
P577
2015-04-01T00:00:00Z