Mobilization of natural killer cells inhibits development of collagen-induced arthritis. - Wikidata - awgldk - TriplyDB

Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

http://www.wikidata.org/entity/Q35197914

about

Cytotoxicity of CD56(bright) NK cells towards autologous activated CD4+ T cells is mediated through NKG2D, LFA-1 and TRAIL and dampened via CD94/NKG2A DNAM-1 control of natural killer cells functions through nectin and nectin-like proteins.NK cells and their ability to modulate T cells during virus infections A p85α-osteopontin axis couples the receptor ICOS to sustained Bcl-6 expression by follicular helper and regulatory T cells.Involvement of RORγt-overexpressing T cells in the development of autoimmune arthritis in mice.Natural killer cells regulate T cell immune responses in primary biliary cirrhosis.NK cells controlling virus-specific T cells: Rheostats for acute vs. persistent infections.Amelioration of arthritis through mobilization of peptide-specific CD8+ regulatory T cells.Effects of anti-NKG2A antibody administration on leukemia and normal hematopoietic cells Natural killer cells in human autoimmune disorders Mechanisms of tissue damage in arthritis.NK cell-based cancer immunotherapy: from basic biology to clinical application.Balance between activating NKG2D, DNAM-1, NKp44 and NKp46 and inhibitory CD94/NKG2A receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts.Up-regulation of NKG2A inhibitory receptor on circulating NK cells contributes to transfusion-induced immunodepression in patients with β-thalassemia major.Host Expression of the CD8 Treg/NK Cell Restriction Element Qa-1 is Dispensable for Transplant Tolerance NKG2D and DNAM-1 activating receptors and their ligands in NK-T cell interactions: role in the NK cell-mediated negative regulation of T cell responses The Role of Microglia and Macrophages in CNS Homeostasis, Autoimmunity, and Cancer.Polymorphisms within the human leucocyte antigen-E gene and their associations with susceptibility to rheumatoid arthritis as well as clinical outcome of anti-tumour necrosis factor therapy.Functional anti-CD94/NKG2A and anti-CD94/NKG2C autoantibodies in patients with systemic lupus erythematosus.Natural killer (NK) cell deficit in coronary artery disease: no aberrations in phenotype but sustained reduction of NK cells is associated with low-grade inflammation.Weak vaccinia virus-induced NK cell regulation of CD4 T cells is associated with reduced NK cell differentiation and cytolytic activity.Natural Killer Cells Regulate Th17 Cells After Autologous Hematopoietic Stem Cell Transplantation for Relapsing Remitting Multiple Sclerosis.Syndecan 4 Regulation of the Development of Autoimmune Arthritis in Mice by Modulating B Cell Migration and Germinal Center Formation.

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Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

http://www.wikidata.org/entity/Q35197914

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

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Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

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type

label

Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

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Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

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Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

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Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

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PNAS.1112188108

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Mobilization of natural killer cells inhibits development of collagen-induced arthritis.

@en

P2093

Carola Schellack Wenander

http://www.w3.org/2001/XMLSchema#string

Harvey Cantor

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Jianmei W Leavenworth

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Pieter Spee

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P2860

Follicular helper CD4 T cells (TFH)

Analysis of the cellular mechanism underlying inhibition of EAE after treatment with anti-NKG2A F(ab')2

New drugs for the treatment of rheumatoid arthritis.

Mechanisms underlying lineage commitment and plasticity of helper CD4+ T cells

CD8+ T regulatory cells express the Ly49 Class I MHC receptor and are defective in autoimmune prone B6-Yaa mice.

Epidemiological studies in incidence, prevalence, mortality, and comorbidity of the rheumatic diseases.

Regulation of activated CD4+ T cells by NK cells via the Qa-1-NKG2A inhibitory pathway.

Follicular B helper T cells in antibody responses and autoimmunity.

Type II collagen autoimmunity in a mouse model of human rheumatoid arthritis.

The role of collagen antibodies in mediating arthritis.

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10.1073/PNAS.1112188108

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108

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