Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
about
Biomarkers of acute kidney injury and associations with short- and long-term outcomesThe cell cycle biomarkers: promising research, but do not oversell themRedox control of senescence and age-related diseasePromoting Kidney Function Recovery in Patients with AKI Requiring RRT.Melatonin promoted renal regeneration in folic acid-induced acute kidney injury via inhibiting nucleocytoplasmic translocation of HMGB1 in tubular epithelial cells.Specific expression of heme oxygenase-1 by myeloid cells modulates renal ischemia-reperfusion injury.Increased expression of p21WAF1/CIP1 in kidney proximal tubules mediates fibrosis.The Role of M2 Macrophages in the Progression of Chronic Kidney Disease following Acute Kidney Injury.Simultaneous deletion of Bax and Bak is required to prevent apoptosis and interstitial fibrosis in obstructive nephropathyBlocking TGF-β Signaling Pathway Preserves Mitochondrial Proteostasis and Reduces Early Activation of PDGFRβ+ Pericytes in Aristolochic Acid Induced Acute Kidney Injury in Wistar Male Rats.Epithelial-to-mesenchymal transition induces cell cycle arrest and parenchymal damage in renal fibrosis.Meclizine Preconditioning Protects the Kidney Against Ischemia-Reperfusion Injury.Activated Protein C Ameliorates Renal Ischemia-Reperfusion Injury by Restricting Y-Box Binding Protein-1 UbiquitinationHigh urinary excretion of kidney injury molecule-1 predicts adverse outcomes in acute kidney injury: a case control study.Atg5-mediated autophagy deficiency in proximal tubules promotes cell cycle G2/M arrest and renal fibrosisFKBP14 overexpression contributes to osteosarcoma carcinogenesis and indicates poor survival outcomeCell cycle control in the kidney.Macrophage Migration Inhibitory Factor Limits Renal Inflammation and Fibrosis by Counteracting Tubular Cell Cycle Arrest.Renal Aging: Causes and Consequences.Cellular senescence in renal ageing and disease.Acute Kidney Injury in Elderly Patients With Chronic Kidney Disease: Do Angiotensin-Converting Enzyme Inhibitors Carry a Risk?D-serine, a novel uremic toxin, induces senescence in human renal tubular cells via GCN2 activation.The proteasome inhibitor bortezomib attenuates renal fibrosis in mice via the suppression of TGF-β1.Adipose-derived mesenchymal stem cells employed exosomes to attenuate AKI-CKD transition through tubular epithelial cell dependent Sox9 activation.Zebrafish Renal Pathology: Emerging Models of Acute Kidney Injury.Exosomes to the Rescue.Long non-coding RNA CHCHD4P4 promotes epithelial-mesenchymal transition and inhibits cell proliferation in calcium oxalate-induced kidney damage.Troy/TNFRSF19 marks epithelial progenitor cells during mouse kidney development that continue to contribute to turnover in adult kidney.Renal replacement in 2050: from renal support to renal replacement?Connective tissue growth factor induces renal fibrosis via epidermal growth factor receptor activation.Acute Kidney Injury as a Condition of Renal Senescence.Targeting histone methyltransferase enhancer of zeste homolog-2 inhibits renal epithelial-mesenchymal transition and attenuates renal fibrosis.
P2860
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P2860
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@ast
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@en
type
label
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@ast
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@en
prefLabel
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@ast
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@en
P2860
P356
P1476
Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury.
@en
P2093
Guillaume Canaud
Joseph V Bonventre
P2860
P304
P356
10.1093/NDT/GFU230
P407
P577
2014-07-12T00:00:00Z