Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats
about
Traumatic Axonal Injury: Mechanisms and Translational OpportunitiesInsights into the metabolic response to traumatic brain injury as revealed by (13)C NMR spectroscopyMitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategiesMolecular mechanisms of cognitive dysfunction following traumatic brain injuryKetone bodies mediate antiseizure effects through mitochondrial permeability transition.Traumatic Brain Injury Increases Cortical Glutamate Network Activity by Compromising GABAergic ControlStandardized bioenergetic profiling of adult mouse cardiomyocytes.Selective Inhibition of the Mitochondrial Permeability Transition Pore Protects against Neurodegeneration in Experimental Multiple SclerosisKnockout of Cyclophilin-D Provides Partial Amelioration of Intrinsic and Synaptic Properties Altered by Mild Traumatic Brain Injury.Altered Cerebellar Circuitry following Thoracic Spinal Cord Injury in Adult Rats.PPAR agonists as therapeutics for CNS trauma and neurological diseasesExogenous BDNF Increases Mitochondrial pCREB and Alleviates Neuronal Metabolic Defects Following Mechanical Injury in a MPTP-Dependent Way.Targeting mitochondrial dysfunction in CNS injury using Methylene Blue; still a magic bullet?N-acetylcysteine amide confers neuroprotection, improves bioenergetics and behavioral outcome following TBI.Traumatic brain injury-induced axonal phenotypes react differently to treatment.Therapeutic efficacy of cyclosporin A against spinal cord injury in rats with hyperglycemia.Post-Injury Treatment with NIM811 Promotes Recovery of Function in Adult Female Rats after Spinal Cord Contusion: A Dose-Response Study.CONTINUOUS INFUSION OF PHENELZINE, CYCLOSPORINE A OR THE COMBINATION: EVALUATION OF MITOCHONDRIAL BIOENERGETICS, OXIDATIVE DAMAGE AND CYTOSKELETAL DEGRADATION FOLLOWING SEVERE CONTROLLED CORTICAL IMPACT TRAUMATIC BRAIN INJURY IN RATS.Striatal Mitochondrial Disruption following Severe Traumatic Brain Injury.Advanced and High-Throughput Method for Mitochondrial Bioenergetics Evaluation in Neurotrauma.Synaptic Mitochondria Sustain More Damage than Non-Synaptic Mitochondria after Traumatic Brain Injury and Are Protected by Cyclosporine A.Variation in PPP3CC Genotype Is Associated with Long-Term Recovery after Severe Brain Injury.SS-31 Provides Neuroprotection by Reversing Mitochondrial Dysfunction after Traumatic Brain InjuryTargeting the mitochondrial permeability transition pore in traumatic central nervous system injury
P2860
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P2860
Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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name
Post-injury administration of ...... traumatic brain injury in rats
@ast
Post-injury administration of ...... traumatic brain injury in rats
@en
type
label
Post-injury administration of ...... traumatic brain injury in rats
@ast
Post-injury administration of ...... traumatic brain injury in rats
@en
prefLabel
Post-injury administration of ...... traumatic brain injury in rats
@ast
Post-injury administration of ...... traumatic brain injury in rats
@en
P2093
P2860
P356
P1476
Post-injury administration of ...... traumatic brain injury in rats
@en
P2093
James R Pauly
Jignesh D Pandya
Joseph E Springer
Melanie L McEwen
Patrick G Sullivan
Ryan D Readnower
P2860
P304
P356
10.1089/NEU.2011.1755
P577
2011-08-29T00:00:00Z