Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia. - Wikidata - awgldk - TriplyDB

Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

http://www.wikidata.org/entity/Q35219857

about

Cell elasticity determines macrophage function M-CSF Mediates Host Defense during Bacterial Pneumonia by Promoting the Survival of Lung and Liver Mononuclear Phagocytes.TLR-mediated inflammatory responses to Streptococcus pneumoniae are highly dependent on surface expression of bacterial lipoproteins Myeloid ZFP36L1 does not regulate inflammation or host defense in mouse models of acute bacterial infection Dynamics of lung defense in pneumonia: resistance, resilience, and remodeling.E-prostanoid 2 receptor signaling suppresses lung innate immunity against Streptococcus pneumoniae.NOS1-derived nitric oxide promotes NF-κB transcriptional activity through inhibition of suppressor of cytokine signaling-1.IκBζ Regulates Human Monocyte Pro-Inflammatory Responses Induced by Streptococcus pneumoniae.Type I alveolar epithelial cells mount innate immune responses during pneumococcal pneumonia.Altered regulation of Toll-like receptor responses impairs antibacterial immunity in the allergic lung Respiratory infection and the impact of pulmonary immunity on lung health and disease No involvement of alveolar macrophages in the initiation of carbon nanoparticle induced acute lung inflammation in mice.Roles of lung epithelium in neutrophil recruitment during pneumococcal pneumonia L-plastin is essential for alveolar macrophage production and control of pulmonary pneumococcal infection Multidimensional pooled shRNA screens in human THP-1 cells identify candidate modulators of macrophage polarization.Myeloid-epithelial cross talk coordinates synthesis of the tissue-protective cytokine leukemia inhibitory factor during pneumonia.Lentiviral delivery of RNAi for in vivo lineage-specific modulation of gene expression in mouse lung macrophages.Regionally compartmentalized resident memory T cells mediate naturally acquired protection against pneumococcal pneumonia.The capacity of pneumococci to activate macrophage NF-kB determines necroptosis and pneumonia severity.MicroRNAs Constitute a Negative Feedback Loop in Streptococcus pneumoniae-Induced Macrophage Activation.THP-1-derived macrophages render lung epithelial cells hypo-responsive to Legionella pneumophila - a systems biology study.Expression of Piwi protein MIWI2 defines a distinct population of multiciliated cells.Embryonic Lethality and Host Immunity of RelA-Deficient Mice Are Mediated by Both Apoptosis and Necroptosis.

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P2860

Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

http://www.wikidata.org/entity/Q35219857

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

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Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

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type

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Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

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Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

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Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

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Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

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RCMB.2010-0210OC

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American Journal of Respiratory Cell and Molecular Biology

P1476

Earliest innate immune responses require macrophage RelA during pneumococcal pneumonia.

@en

P2093

Bryanne E Robson

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J Daniel Ferrari

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Kazuko Yamamoto

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Lynnelle A Pittet

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Roland M Schmid

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P2860

Lung infection--a public health priority

Secretion without Golgi

Interleukin-17 and type 17 helper T cells

Integrating cell-signalling pathways with NF-kappaB and IKK function

Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B

Differential activation of inflammatory pathways in A549 type II pneumocytes by Streptococcus pneumoniae strains with different adherence properties.

CXC chemokine receptor CXCR2 is essential for protective innate host response in murine Pseudomonas aeruginosa pneumonia.

Granulocyte apoptosis and its role in the resolution and control of lung inflammation.

Importance of CXC chemokine receptor 2 in alveolar neutrophil and exudate macrophage recruitment in response to pneumococcal lung infection.

Illustration of pneumococcal polysaccharide capsule during adherence and invasion of epithelial cells.

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573-581

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10.1165/RCMB.2010-0210OC

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3

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45

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Joseph P Mizgerd

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2011-01-07T00:00:00Z

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21216972

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3175578

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