Vascular dysfunction in experimental diabetes is improved by pentaerithrityl tetranitrate but not isosorbide-5-mononitrate therapy. - Wikidata - awgldk - TriplyDB

Vascular dysfunction in experimental diabetes is improved by pentaerithrityl tetranitrate but not isosorbide-5-mononitrate therapy.

Vascular dysfunction in experimental diabetes is improved by pentaerithrityl tetranitrate but not isosorbide-5-mononitrate therapy.

http://www.wikidata.org/entity/Q35227188

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Organic Nitrate Therapy, Nitrate Tolerance, and Nitrate-Induced Endothelial Dysfunction: Emphasis on Redox Biology and Oxidative Stress Exploiting the Pleiotropic Antioxidant Effects of Established Drugs in Cardiovascular Disease Mitochondrial redox signaling: Interaction of mitochondrial reactive oxygen species with other sources of oxidative stress Enhanced NF-κB activity impairs vascular function through PARP-1-, SP-1-, and COX-2-dependent mechanisms in type 2 diabetes.European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Review article: the role of oxidative stress in pathogenesis and treatment of inflammatory bowel diseases.Renal response to L-arginine in diabetic rats. A possible link between nitric oxide system and aquaporin-2 Nitroglycerin tolerance in caveolin-1 deficient mice.Glutathionylation mediates angiotensin II-induced eNOS uncoupling, amplifying NADPH oxidase-dependent endothelial dysfunction The sodium-glucose co-transporter 2 inhibitor empagliflozin improves diabetes-induced vascular dysfunction in the streptozotocin diabetes rat model by interfering with oxidative stress and glucotoxicity Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair.Direct Antioxidant Properties of Bilirubin and Biliverdin. Is there a Role for Biliverdin Reductase?β3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade.Regulation of endothelial nitric-oxide synthase (NOS) S-glutathionylation by neuronal NOS: evidence of a functional interaction between myocardial constitutive NOS isoforms.β3 Adrenergic Stimulation Restores Nitric Oxide/Redox Balance and Enhances Endothelial Function in Hyperglycemia.Nuclear factor kappa B inhibition improves conductance artery function in type 2 diabetic mice Protein tyrosine nitration and thiol oxidation by peroxynitrite-strategies to prevent these oxidative modifications Redox modulation of endothelial nitric oxide synthase by glutaredoxin-1 through reversible oxidative post-translational modification.Molecular mechanisms of the crosstalk between mitochondria and NADPH oxidase through reactive oxygen species-studies in white blood cells and in animal models.Efficacy of the long-acting nitro vasodilator pentaerithrityl tetranitrate in patients with chronic stable angina pectoris receiving anti-anginal background therapy with beta-blockers: a 12-week, randomized, double-blind, placebo-controlled trial.Pentaerythritol Tetranitrate In Vivo Treatment Improves Oxidative Stress and Vascular Dysfunction by Suppression of Endothelin-1 Signaling in Monocrotaline-Induced Pulmonary Hypertension.Free radical biology of the cardiovascular system.Prevention of diabetic complications by activation of Nrf2: diabetic cardiomyopathy and nephropathy.Emerging drugs for managing kidney disease in patients with diabetes.Crosstalk of mitochondria with NADPH oxidase via reactive oxygen and nitrogen species signalling and its role for vascular function.Reversibility of endothelial dysfunction in diabetes: role of polyphenols.Taking up the cudgels for the traditional reactive oxygen and nitrogen species detection assays and their use in the cardiovascular system.Glycemic Control with Ipragliflozin, a Novel Selective SGLT2 Inhibitor, Ameliorated Endothelial Dysfunction in Streptozotocin-Induced Diabetic Mouse Mechanistic link between erectile dysfunction and systemic endothelial dysfunction in type 2 diabetic rats.Heme oxygenase-1 induction and organic nitrate therapy: beneficial effects on endothelial dysfunction, nitrate tolerance, and vascular oxidative stress Chronic therapy with isosorbide-5-mononitrate causes endothelial dysfunction, oxidative stress, and a marked increase in vascular endothelin-1 expression.Oxidative Stress in Atherosclerosis.Serum asymmetric dimethylarginine and arginine levels predict microvascular and macrovascular complications in type 2 diabetes mellitus.

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P2860

Vascular dysfunction in experimental diabetes is improved by pentaerithrityl tetranitrate but not isosorbide-5-mononitrate therapy.

http://www.wikidata.org/entity/Q35227188

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2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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Vascular dysfunction in experi ...... sorbide-5-mononitrate therapy.

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Andreas Daiber

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Christian Otto

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Eberhard Schulz

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Franziska Bollmann

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Hartmut Kleinert

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Maike Knorr

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Matthias Oelze

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Michael Hausding

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P2860

Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation

Intensive Diabetes Treatment and Cardiovascular Disease in Patients with Type 1 Diabetes

The nitric oxide donor pentaerythritol tetranitrate can preserve endothelial function in established atherosclerosis

Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly

Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

Endothelial protection by pentaerithrityl trinitrate: bilirubin and carbon monoxide as possible mediators.

Heme oxygenase-1 induction may explain the antioxidant profile of pentaerythrityl trinitrate.

The antioxidant defense protein ferritin is a novel and specific target for pentaerithrityl tetranitrate in endothelial cells.

Mechanisms underlying endothelial dysfunction in diabetes mellitus.

Mechanisms underlying recoupling of eNOS by HMG-CoA reductase inhibition in a rat model of streptozotocin-induced diabetes mellitus.

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