Novel endogenous N-acyl amides activate TRPV1-4 receptors, BV-2 microglia, and are regulated in brain in an acute model of inflammation.

Novel endogenous N-acyl amides activate TRPV1-4 receptors, BV-2 microglia, and are regulated in brain in an acute model of inflammation.

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http://www.wikidata.org/entity/Q35227731
TRPV1 channels are critical brain inflammation detectors and neuropathic pain biomarkers in mice.Attenuation of persistent pain-related behavior by fatty acid amide hydrolase (FAAH) inhibitors in a rat model of HIV sensory neuropathy.Lipopolysaccharide-induced murine embryonic resorption involves changes in endocannabinoid profiling and alters progesterone secretion and inflammatory response by a CB1-mediated fashion.A pro-nociceptive phenotype unmasked in mice lacking fatty-acid amide hydrolaseLevels of bioactive lipids in cooking oils: olive oil is the richest source of oleoyl serine.Affinin (Spilanthol), Isolated from Heliopsis longipes, Induces Vasodilation via Activation of Gasotransmitters and Prostacyclin Signaling Pathways.Transient Receptor Potential Channels in Microglia: Roles in Physiology and Disease.Lipidomics profile of a NAPE-PLD KO mouse provides evidence of a broader role of this enzyme in lipid metabolism in the brain.Broad impact of deleting endogenous cannabinoid hydrolyzing enzymes and the CB1 cannabinoid receptor on the endogenous cannabinoid-related lipidome in eight regions of the mouse brain.Where's my entourage? The curious case of 2-oleoylglycerol, 2-linolenoylglycerol, and 2-palmitoylglycerolThe truth in complexes: perspectives on ion channel signaling nexuses in the nervous system.Targeting fatty acid amide hydrolase and transient receptor potential vanilloid-1 simultaneously to modulate colonic motility and visceral sensation in the mouse: A pharmacological intervention with N-arachidonoyl-serotonin (AA-5-HT).Induction of chronic migraine phenotypes in a rat model after environmental irritant exposure.Δ9-Tetrahydrocannabinol changes the brain lipidome and transcriptome differentially in the adolescent and the adult.Microglia in Alzheimer's Disease: A Role for Ion Channels
P2860

Novel endogenous N-acyl amides activate TRPV1-4 receptors, BV-2 microglia, and are regulated in brain in an acute model of inflammation.

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http://www.wikidata.org/entity/Q35227731
description
2014 nî lūn-bûn
@nan
2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@ast
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@en
type
label
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@ast
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@en
prefLabel
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@ast
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P478
P50
P577
P5875
P698
P921
P932
P356
P1433
P1476
Novel endogenous N-acyl amides ...... n acute model of inflammation.
@en
P2093
P2860
P304
P31
P356
P478
P50
P577
P5875
P698
P921
P932