IL-17 and IFN-gamma mediate the elicitation of contact hypersensitivity responses by different mechanisms and both are required for optimal responses.
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Eosinophil-dependent skin innervation and itching following contact toxicant exposure in mice.Novel concept of iSALT (inducible skin-associated lymphoid tissue) in the elicitation of allergic contact dermatitisPraziquantel facilitates IFN-γ-producing CD8+ T cells (Tc1) and IL-17-producing CD8+ T cells (Tc17) responses to DNA vaccination in miceAnti-inflammatory potential of Capparis spinosa L. in vivo in mice through inhibition of cell infiltration and cytokine gene expressionToll-like receptor-4 deficiency enhances repair of UVR-induced cutaneous DNA damage by nucleotide excision repair mechanism.Differential Effects of Naja naja atra Venom on Immune Activity.Secretion of IFN-gamma but not IL-17 by CD1d-restricted NKT cells enhances rejection of skin grafts expressing epithelial cell-derived antigen.IL-17 promotes tumor development through the induction of tumor promoting microenvironments at tumor sites and myeloid-derived suppressor cellsPreventive and therapeutic potential of placental extract in contact hypersensitivitySM934 treated lupus-prone NZB × NZW F1 mice by enhancing macrophage interleukin-10 production and suppressing pathogenic T cell development.Host defense mechanisms in secondary syphilitic lesions: a role for IFN-gamma-/IL-17-producing CD8+ T cells?Dual functions of prostaglandin D2 in murine contact hypersensitivity via DP and CRTH2.IL-1 receptor signaling is required at multiple stages of sensitization and elicitation of the contact hypersensitivity responseDermal γδ T Cells Do Not Freely Re-Circulate Out of Skin and Produce IL-17 to Promote Neutrophil Infiltration during Primary Contact HypersensitivityIn Vivo Suppression of Heat Shock Protein (HSP)27 and HSP70 Accelerates DMBA-Induced Skin Carcinogenesis by Inducing Antigenic Unresponsiveness to the Initiating Carcinogenic Chemical.Transcription factor NFAT1 controls allergic contact hypersensitivity through regulation of activation induced cell death program.ASK1 promotes the contact hypersensitivity response through IL-17 production.Diagnostic approach in allergic and irritant contact dermatitis.Immunological mechanisms of contact hypersensitivity in mice.Inflammatory and immune mechanisms in contact hypersensitivity (CHS) in rats.Natural killer cells: walking three paths down memory lane.TH17 is involved in the remarkable regression of metastatic malignant melanoma to topical diphencyprone.Natural (Mineral, Vegetable, Coconut, Essential) Oils and Contact Dermatitis.Deletion of cannabinoid receptors 1 and 2 exacerbates APC function to increase inflammation and cellular immunity during influenza infectionEmerging Skin T-Cell Functions in Response to Environmental InsultsHmgb1-IL-23-IL-17-IL-6-Stat3 axis promotes tumor growth in murine models of melanoma.Interleukin-17 mediated inflammatory responses are required for ultraviolet radiation-induced immune suppression.Systemic immunogenicity of para-Phenylenediamine and Diphenylcyclopropenone: two potent contact allergy-inducing haptens.1,25-dihydroxyvitamin D3 enhances the ability of transferred CD4+ CD25+ cells to modulate T helper type 2-driven asthmatic responses.Aquaporin-9-expressing neutrophils are required for the establishment of contact hypersensitivity.[Immunology of contact allergy].Paeoniflorin inhibits inflammatory responses in mice with allergic contact dermatitis by regulating the balance between inflammatory and anti-inflammatory cytokines.Topical administration of the pan-Src kinase inhibitors, dasatinib and LCB 03-0110, prevents allergic contact dermatitis in mice.Natural killer cell-mediated contact sensitivity develops rapidly and depends on interferon-α, interferon-γ and interleukin-12.Rapid allergen-induced interleukin-17 and interferon-γ secretion by skin-resident memory CD8+ T cells.CXCR5-Overexpressing Mesenchymal Stromal Cells Exhibit Enhanced Homing and Can Decrease Contact Hypersensitivity.Impact of the magnitude of sensitization dose on the incidence and intensity of CHS to dinitrochlorobenzene (DNCB): insight from ear swelling and challenged-skin draining lymph node response in rats.Analysis of the inflammatory response in HY-TCR transgenic mice highlights the pathogenic potential of CD4- CD8- T cells.Attenuation of contact hypersensitivity by cell-permeable heat shock protein 70 in BALB/c mouse model.Innate immunity and effector and regulatory mechanisms involved in allergic contact dermatitis.
P2860
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P2860
IL-17 and IFN-gamma mediate the elicitation of contact hypersensitivity responses by different mechanisms and both are required for optimal responses.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@ast
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@en
type
label
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@ast
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@en
prefLabel
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@ast
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@en
P2093
P2860
P356
P1476
IL-17 and IFN-gamma mediate th ...... equired for optimal responses.
@en
P2093
P2860
P304
P356
10.4049/JIMMUNOL.0804108
P407
P577
2009-06-24T00:00:00Z