Gln3 phosphorylation and intracellular localization in nutrient limitation and starvation differ from those generated by rapamycin inhibition of Tor1/2 in Saccharomyces cerevisiae.
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The role of autophagy in genome stability through suppression of abnormal mitosis under starvationAlterations in the Ure2 αCap domain elicit different GATA factor responses to rapamycin treatment and nitrogen limitationStb3 plays a role in the glucose-induced transition from quiescence to growth in Saccharomyces cerevisiae.Differential regulation and substrate preferences in two peptide transporters of Saccharomyces cerevisiae.Mds3 regulates morphogenesis in Candida albicans through the TOR pathwayConstitutive and nitrogen catabolite repression-sensitive production of Gat1 isoformsGene discovery and transcript analyses in the corn smut pathogen Ustilago maydis: expressed sequence tag and genome sequence comparison.A domain in the transcription activator Gln3 specifically required for rapamycin responsiveness.Distinct phosphatase requirements and GATA factor responses to nitrogen catabolite repression and rapamycin treatment in Saccharomyces cerevisiae.The transduction of the nitrogen regulation signal in Saccharomyces cerevisiae.State transitions in the TORC1 signaling pathway and information processing in Saccharomyces cerevisiaeActin cytoskeleton is required for nuclear accumulation of Gln3 in response to nitrogen limitation but not rapamycin treatment in Saccharomyces cerevisiae.Regulation of amino acid, nucleotide, and phosphate metabolism in Saccharomyces cerevisiae.Discovery of parasite virulence genes reveals a unique regulator of chromosome condensation 1 ortholog critical for efficient nuclear trafficking.Tor Signaling Regulates Transcription of Amino Acid Permeases through a GATA Transcription Factor Gaf1 in Fission YeastThe TOR complex 1 is a direct target of Rho1 GTPase.GATA Factor Regulation in Excess Nitrogen Occurs Independently of Gtr-Ego Complex-Dependent TorC1 Activation.Nuclear Gln3 Import Is Regulated by Nitrogen Catabolite Repression Whereas Export Is Specifically Regulated by Glutaminegln3 mutations dissociate responses to nitrogen limitation (nitrogen catabolite repression) and rapamycin inhibition of TorC1Vesicular Trafficking Systems Impact TORC1-Controlled Transcriptional Programs in Saccharomyces cerevisiaeNutritional control via Tor signaling in Saccharomyces cerevisiae.Nuclear translocation of Gln3 in response to nutrient signals requires Golgi-to-endosome trafficking in Saccharomyces cerevisiaeRapamycin bypasses vesicle-mediated signaling events to activate Gln3 in Saccharomyces cerevisiae.Formalin can alter the intracellular localization of some transcription factors in Saccharomyces cerevisiaeNitrogen catabolite repression-sensitive transcription as a readout of Tor pathway regulation: the genetic background, reporter gene and GATA factor assayed determine the outcomes.Recent advances in nitrogen regulation: a comparison between Saccharomyces cerevisiae and filamentous fungiFive conditions commonly used to down-regulate tor complex 1 generate different physiological situations exhibiting distinct requirements and outcomes.Signaling cascades as drug targets in model and pathogenic fungi.Intranuclear function for protein phosphatase 2A: Pph21 and Pph22 are required for rapamycin-induced GATA factor binding to the DAL5 promoter in yeastMolecular communication: crosstalk between the Snf1 and other signaling pathways.Nitrogen-responsive regulation of GATA protein family activators Gln3 and Gat1 occurs by two distinct pathways, one inhibited by rapamycin and the other by methionine sulfoximine.Multiple Targets on the Gln3 Transcription Activator Are Cumulatively Required for Control of Its Cytoplasmic Sequestration.Differing responses of Gat1 and Gln3 phosphorylation and localization to rapamycin and methionine sulfoximine treatment in Saccharomyces cerevisiae.Ammonia-specific regulation of Gln3 localization in Saccharomyces cerevisiae by protein kinase Npr1.Nuclear accumulation of the GATA factor AreA in response to complete nitrogen starvation by regulation of nuclear exportMethionine sulfoximine treatment and carbon starvation elicit Snf1-independent phosphorylation of the transcription activator Gln3 in Saccharomyces cerevisiae.In vivo specificity of Ure2 protection from heavy metal ion and oxidative cellular damage in Saccharomyces cerevisiae.Synergistic operation of four cis-acting elements mediate high level DAL5 transcription in Saccharomyces cerevisiae.Saccharomyces cerevisiae Sit4 phosphatase is active irrespective of the nitrogen source provided, and Gln3 phosphorylation levels become nitrogen source-responsive in a sit4-deleted strain.Stress-responsive Gln3 localization in Saccharomyces cerevisiae is separable from and can overwhelm nitrogen source regulation.
P2860
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P2860
Gln3 phosphorylation and intracellular localization in nutrient limitation and starvation differ from those generated by rapamycin inhibition of Tor1/2 in Saccharomyces cerevisiae.
description
2003 nî lūn-bûn
@nan
2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@ast
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@en
type
label
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@ast
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@en
prefLabel
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@ast
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@en
P2093
P2860
P356
P1476
Gln3 phosphorylation and intra ...... 2 in Saccharomyces cerevisiae.
@en
P2093
Ajit Kulkarni
Jennifer J Tate
Kathleen H Cox
Terrance G Cooper
P2860
P304
10270-10278
P356
10.1074/JBC.M312023200
P407
P577
2003-12-16T00:00:00Z