Activation of matrix metalloproteinases following anti-Aβ immunotherapy; implications for microhemorrhage occurrence. - Wikidata - awgldk - TriplyDB

Activation of matrix metalloproteinases following anti-Aβ immunotherapy; implications for microhemorrhage occurrence.

Activation of matrix metalloproteinases following anti-Aβ immunotherapy; implications for microhemorrhage occurrence.

http://www.wikidata.org/entity/Q35240865

about

Arginine deprivation and immune suppression in a mouse model of Alzheimer's disease Cerebral microhemorrhages: mechanisms, consequences, and prevention.A coimmunization vaccine of Aβ42 ameliorates cognitive deficits without brain inflammation in an Alzheimer's disease model.Induction of hyperhomocysteinemia models vascular dementia by induction of cerebral microhemorrhages and neuroinflammation.Roles of NFκB-miR-29s-MMP-2 circuitry in experimental choroidal neovascularization.Aging mesenchymal stem cells fail to protect because of impaired migration and antiinflammatory response.β-amyloid deposition is shifted to the vasculature and memory impairment is exacerbated when hyperhomocysteinemia is induced in APP/PS1 transgenic mice.Dietary methanol regulates human gene activity The impact of human and mouse differences in NOS2 gene expression on the brain's redox and immune environment.A lipoprotein receptor cluster IV mutant preferentially binds amyloid-β and regulates its clearance from the mouse brain Diverse inflammatory responses in transgenic mouse models of Alzheimer's disease and the effect of immunotherapy on these responses.MicroRNA-195a-3p inhibits angiogenesis by targeting Mmp2 in murine mesenchymal stem cells.Unfractionated bone marrow cells attenuate paraquat-induced glomerular injury and acute renal failure by modulating the inflammatory response.Nitric oxide-mediated regulation of β-amyloid clearance via alterations of MMP-9/TIMP-1 Reduced Efficacy of Anti-Aβ Immunotherapy in a Mouse Model of Amyloid Deposition and Vascular Cognitive Impairment Comorbidity.Intracranial injection of Gammagard, a human IVIg, modulates the inflammatory response of the brain and lowers Aβ in APP/PS1 mice along a different time course than anti-Aβ antibodies.Imaging of cerebrovascular pathology in animal models of Alzheimer's disease Matrix Metalloproteinase in Blood-Brain Barrier Breakdown in Dementia.A Novel Small Molecule Modulator of Amyloid Pathology.

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P2860

Activation of matrix metalloproteinases following anti-Aβ immunotherapy; implications for microhemorrhage occurrence.

http://www.wikidata.org/entity/Q35240865

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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Activation of matrix metallopr ...... or microhemorrhage occurrence.

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Activation of matrix metallopr ...... or microhemorrhage occurrence.

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Activation of matrix metallopr ...... or microhemorrhage occurrence.

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Activation of matrix metallopr ...... or microhemorrhage occurrence.

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Activation of matrix metallopr ...... or microhemorrhage occurrence.

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Journal of Neuroinflammation

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Activation of matrix metallopr ...... or microhemorrhage occurrence.

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Dave Morgan

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David A Wink

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Donna M Wilcock

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Lisa A Ridnour

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Marcia N Gordon

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Tiffany L Taylor

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Passive immunotherapy against Abeta in aged APP-transgenic mice reverses cognitive deficits and depletes parenchymal amyloid deposits in spite of increased vascular amyloid and microhemorrhage

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Matrix metalloproteinases

The tumor necrosis factor-alpha converting enzyme (TACE): a unique metalloproteinase with highly defined substrate selectivity

Increased gelatinase A (MMP-2) and gelatinase B (MMP-9) activities in human brain after focal ischemia

Alzheimer's disease: the amyloid cascade hypothesis

Matrix metalloproteinase-9 degrades amyloid-beta fibrils in vitro and compact plaques in situ.

Neuropathology and amyloid-β spectrum in a bapineuzumab immunotherapy recipient.

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