PITX2 is involved in stress response in cultured human trabecular meshwork cells through regulation of SLC13A3. - Wikidata - awgldk - TriplyDB

PITX2 is involved in stress response in cultured human trabecular meshwork cells through regulation of SLC13A3.

PITX2 is involved in stress response in cultured human trabecular meshwork cells through regulation of SLC13A3.

http://www.wikidata.org/entity/Q35247724

about

GPR158, an orphan member of G protein-coupled receptor Family C: glucocorticoid-stimulated expression and novel nuclear role The Role of INDY in Metabolic Regulation Strain-Dependent Anterior Segment Dysgenesis and Progression to Glaucoma in Col4a1 Mutant Mice.PITX2c loss-of-function mutations responsible for congenital atrial septal defects.Prevalence and spectrum of PITX2c mutations associated with congenital heart disease.PITX2C loss-of-function mutations responsible for idiopathic atrial fibrillation.Sodium-coupled dicarboxylate and citrate transporters from the SLC13 family.Ion transport in the zebrafish kidney from a human disease angle: possibilities, considerations, and future perspectives.Single stranded adeno-associated virus achieves efficient gene transfer to anterior segment in the mouse eye.

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P2860

PITX2 is involved in stress response in cultured human trabecular meshwork cells through regulation of SLC13A3.

http://www.wikidata.org/entity/Q35247724

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

PITX2 is involved in stress re ...... through regulation of SLC13A3.

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PITX2 is involved in stress re ...... through regulation of SLC13A3.

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PITX2 is involved in stress re ...... through regulation of SLC13A3.

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PITX2 is involved in stress re ...... through regulation of SLC13A3.

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Investigative Ophthalmology & Visual Science

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PITX2 is involved in stress re ...... through regulation of SLC13A3.

@en

P2093

Fred B Berry

http://www.w3.org/2001/XMLSchema#string

M Hermina Strungaru

http://www.w3.org/2001/XMLSchema#string

Michael A Walter

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Pascal Belleau

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Tim Footz

http://www.w3.org/2001/XMLSchema#string

Vincent Raymond

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Yi Liu

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P2860

Identification of a dominant negative homeodomain mutation in Rieger syndrome

FOXC1 is required for cell viability and resistance to oxidative stress in the eye through the transcriptional regulation of FOXO1A

Functional interactions between FOXC1 and PITX2 underlie the sensitivity to FOXC1 gene dose in Axenfeld-Rieger syndrome and anterior segment dysgenesis

Autosomal dominant iris hypoplasia is caused by a mutation in the Rieger syndrome (RIEG/PITX2) gene

Mutation in the RIEG1 gene in patients with iridogoniodysgenesis syndrome

Proteomic and genomic analysis of PITX2 interacting and regulating networks

Global data on visual impairment in the year 2002

PITX2 regulates procollagen lysyl hydroxylase (PLOD) gene expression: implications for the pathology of Rieger syndrome

Model-based analysis of oligonucleotide arrays: model validation, design issues and standard error application

Model-based analysis of oligonucleotide arrays: expression index computation and outlier detection

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7625-7633

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10.1167/IOVS.10-6967

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10

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52

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51601691

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21873665

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3183983

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