Protective role of transient pore openings in calcium handling by cardiac mitochondria. - Wikidata - awgldk - TriplyDB

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

http://www.wikidata.org/entity/Q35266787

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Calcium signaling as a mediator of cell energy demand and a trigger to cell death Cell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthase Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.ER-mitochondrial calcium flow underlies vulnerability of mechanosensory hair cells to damage.Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Diversity of mitochondrial Ca²⁺ signaling in rat neonatal cardiomyocytes: evidence from a genetically directed Ca²⁺ probe, mitycam-E31Q.Mitochondrial dysfunction in uremic cardiomyopathy.Harnessing the Power of Integrated Mitochondrial Biology and Physiology: A Special Report on the NHLBI Mitochondria in Heart Diseases Initiative.Physiological and pathological roles of the mitochondrial permeability transition pore in the heart.Synergistic triggering of superoxide flashes by mitochondrial Ca2+ uniport and basal reactive oxygen species elevation.Individual Cardiac Mitochondria Undergo Rare Transient Permeability Transition Pore Openings.Ischemia/Reperfusion injury protection by mesenchymal stem cell derived antioxidant capacity.Massive endocytosis triggered by surface membrane palmitoylation under mitochondrial control in BHK fibroblasts.Mitochondrial channels: ion fluxes and more.Hexokinases and cardioprotection.New Roles for an Old Pore Mitochondrial delivery of doxorubicin by triphenylphosphonium-functionalized hyperbranched nanocarriers results in rapid and severe cytotoxicity.Impact of caloric restriction on myocardial ischaemia/reperfusion injury and new therapeutic options to mimic its effects Mff-Dependent Mitochondrial Fission Contributes to the Pathogenesis of Cardiac Microvasculature Ischemia/Reperfusion Injury via Induction of mROS-Mediated Cardiolipin Oxidation and HK2/VDAC1 Disassociation-Involved mPTP Opening.Physiological roles of the mitochondrial permeability transition pore.Effects of 915 nm GaAs diode laser on mitochondria of human dermal fibroblasts: analysis with confocal microscopy.

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P2860

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

http://www.wikidata.org/entity/Q35266787

description

2011 nî lūn-bûn

@nan

2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2011年の論文

@ja

2011年論文

@yue

2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

@ast

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

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type

label

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

@ast

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

@en

prefLabel

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

@ast

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

@en

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P356

JBC.M111.239921

P1433

Journal of Biological Chemistry

P1476

Protective role of transient pore openings in calcium handling by cardiac mitochondria.

@en

P2093

James N Weiss

http://www.w3.org/2001/XMLSchema#string

Jun-Hai Yang

http://www.w3.org/2001/XMLSchema#string

Ling Yang

http://www.w3.org/2001/XMLSchema#string

Paavo Korge

http://www.w3.org/2001/XMLSchema#string

Zhilin Qu

http://www.w3.org/2001/XMLSchema#string

P2860

A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter

Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice.

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state

Mitochondrial free calcium regulation during sarcoplasmic reticulum calcium release in rat cardiac myocytes.

Fluctuations in mitochondrial membrane potential caused by repetitive gating of the permeability transition pore

Mitochondrial transport of cations: channels, exchangers, and permeability transition.

Anion carriers in fatty acid-mediated physiological uncoupling.

Imaging the permeability pore transition in single mitochondria.

Role of the mitochondrial permeability transition in myocardial disease.

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34851-34857

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scholarly article

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10.1074/JBC.M111.239921

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40

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286

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3186421

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