BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis - Wikidata - awgldk - TriplyDB

BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

http://www.wikidata.org/entity/Q35287026

about

Interactome of the negative regulator of nuclear import BRCA1-binding protein 2 Research Progress on the Risk Factors and Outcomes of Human Carotid Atherosclerotic Plaques.Lack of association between a functional variant of the BRCA-1 related associated protein (BRAP) gene and ischemic stroke Brap2 regulates temporal control of NF-κB localization mediated by inflammatory response.A two-stage association study suggests BRAP as a susceptibility gene for schizophrenia.Central catalytic domain of BRAP (RNF52) recognizes the types of ubiquitin chains and utilizes oligo-ubiquitin for ubiquitylation.Impact of single nucleotide polymorphism on short stature and reduced tongue pressure among community-dwelling elderly Japanese participants: a cross-sectional study.Premature ovarian aging in BRCA carriers: a prototype of systemic precocious aging?

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

http://www.wikidata.org/entity/Q35287026

description

2011 nî lūn-bûn

@nan

2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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MOLMED.2011.00043

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Molecular Medicine

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BRAP Activates Inflammatory Cascades and Increases the Risk for Carotid Atherosclerosis

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Ku-Chung Chen

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Ming-Hong Chang

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Ming-Lung Yu

http://www.w3.org/2001/XMLSchema#string

Sheng-Hsiung Sheu

http://www.w3.org/2001/XMLSchema#string

Suh-Hang Hank Juo

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Yi-Chu Liao

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Yuh-Cherng Guo

http://www.w3.org/2001/XMLSchema#string

Yung-Song Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Ras regulates assembly of mitogenic signalling complexes through the effector protein IMP

The NF-kappa B and I kappa B proteins: new discoveries and insights

Identification of a novel cytoplasmic protein that specifically binds to nuclear localization signal motifs

Role of unphosphorylated, newly synthesized I kappa B beta in persistent activation of NF-kappa B

Atherosclerosis — An Inflammatory Disease

Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

RelA/p65 regulation of IkappaBbeta.

The IkappaB-NF-kappaB signaling module: temporal control and selective gene activation

Single nucleotide polymorphisms in monocyte chemoattractant protein-1 and its receptor act synergistically to increase the risk of carotid atherosclerosis.

Technology Insight: ultrasound measurement of carotid plaque--patient management, genetic research, and therapy evaluation.

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molmed.2011.00043

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Toshihiro Tanaka

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