Excess cones in the retinal degeneration rd7 mouse, caused by the loss of function of orphan nuclear receptor Nr2e3, originate from early-born photoreceptor precursors
about
Photoreceptor cell fate specification in vertebratesMinireview: the role of nuclear receptors in photoreceptor differentiation and diseaseThe Crystal Structure of the Orphan Nuclear Receptor NR2E3/PNR Ligand Binding Domain Reveals a Dimeric Auto-Repressed ConformationBiology and therapy of inherited retinal degenerative disease: insights from mouse modelsPotential of Small Molecule-Mediated Reprogramming of Rod Photoreceptors to Treat Retinitis PigmentosaNetwork-based bioinformatics analysis of spatio-temporal RNA-Seq data reveals transcriptional programs underpinning normal and aberrant retinal development.Defects in the outer limiting membrane are associated with rosette development in the Nrl-/- retina.Feedback induction of a photoreceptor-specific isoform of retinoid-related orphan nuclear receptor β by the rod transcription factor NRL.Overexpression of retinal degeneration slow (RDS) protein adversely affects rods in the rd7 model of enhanced S-cone syndromeVision from next generation sequencing: multi-dimensional genome-wide analysis for producing gene regulatory networks underlying retinal development, aging and diseaseDifferential dimerization of variants linked to enhanced S-cone sensitivity syndrome (ESCS) located in the NR2E3 ligand-binding domain.S cones: Evolution, retinal distribution, development, and spectral sensitivity.Multimodal Regulation Orchestrates Normal and Complex Disease States in the RetinaBrief report: self-organizing neuroepithelium from human pluripotent stem cells facilitates derivation of photoreceptors.Small molecule Photoregulin3 prevents retinal degeneration in the RhoP23H mouse model of retinitis pigmentosa.Epigenetic control of gene regulation during development and disease: A view from the retina.Two-color pupillometry in enhanced S-cone syndrome caused by NR2E3 mutations.
P2860
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P2860
Excess cones in the retinal degeneration rd7 mouse, caused by the loss of function of orphan nuclear receptor Nr2e3, originate from early-born photoreceptor precursors
description
2011 nî lūn-bûn
@nan
2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Excess cones in the retinal de ...... -born photoreceptor precursors
@ast
Excess cones in the retinal de ...... -born photoreceptor precursors
@en
type
label
Excess cones in the retinal de ...... -born photoreceptor precursors
@ast
Excess cones in the retinal de ...... -born photoreceptor precursors
@en
prefLabel
Excess cones in the retinal de ...... -born photoreceptor precursors
@ast
Excess cones in the retinal de ...... -born photoreceptor precursors
@en
P2860
P356
P1476
Excess cones in the retinal de ...... -born photoreceptor precursors
@en
P2093
Hong Cheng
Naheed W Khan
P2860
P304
P356
10.1093/HMG/DDR334
P577
2011-08-03T00:00:00Z