Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathway - Wikidata - awgldk - TriplyDB

Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathway

Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathway

http://www.wikidata.org/entity/Q35311159

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Sphingolipids and antimicrobial peptides: function and roles in atopic dermatitis Epidermal permeability barrier defects and barrier repair therapy in atopic dermatitis Role of micronutrients in skin health and function Cathelicidin LL-37: an antimicrobial peptide with a role in inflammatory skin disease DNA methylation directly downregulates human cathelicidin antimicrobial peptide gene (CAMP) promoter activity.Endoplasmic reticulum calcium, stress, and cell-to-cell adhesion.Activation of innate immune-response genes in little brown bats (Myotis lucifugus) infected with the fungus Pseudogymnoascus destructans.An endoplasmic reticulum stress-initiated sphingolipid metabolite, ceramide-1-phosphate, regulates epithelial innate immunity by stimulating β-defensin production Boosting innate immunity: development and validation of a cell-based screening assay to identify LL-37 inducers.Synergistic induction of human cathelicidin antimicrobial peptide gene expression by vitamin D and stilbenoids.The dietary ingredient, genistein, stimulates cathelicidin antimicrobial peptide expression through a novel S1P-dependent mechanism.Sphingosine kinase 1 activation enhances epidermal innate immunity through sphingosine-1-phosphate stimulation of cathelicidin production Vitamin C Stimulates Epidermal Ceramide Production by Regulating Its Metabolic Enzymes Curcumin induces human cathelicidin antimicrobial peptide gene expression through a vitamin D receptor-independent pathway Interleukin 13 exposure enhances vitamin D-mediated expression of the human cathelicidin antimicrobial peptide 18/LL-37 in bronchial epithelial cells.A novel role of a lipid species, sphingosine-1-phosphate, in epithelial innate immunity.ER stress stimulates production of the key antimicrobial peptide, cathelicidin, by forming a previously unidentified intracellular S1P signaling complex Predicted vitamin D status and incidence of tooth loss and periodontitis.Resveratrol stimulates sphingosine-1-phosphate signaling of cathelicidin production.Entinostat up-regulates the CAMP gene encoding LL-37 via activation of STAT3 and HIF-1α transcription factors.Little peptide, big effects: the role of LL-37 in inflammation and autoimmune disease Antimicrobial Activity of Mesenchymal Stem Cells: Current Status and New Perspectives of Antimicrobial Peptide-Based Therapies Induction of the human cathelicidin LL-37 as a novel treatment against bacterial infections.Antimicrobial peptide elicitors: new hope for the post-antibiotic era.Endoplasmic reticulum stress: key promoter of rosacea pathogenesis.Cutaneous Immune Defenses Against Staphylococcus aureus Infections.Unique features of human cathelicidin LL-37.PTH and Vitamin D.The Roles of Cathelicidin LL-37 in Inflammatory Bowel Disease.C/EBPα and the Vitamin D Receptor Cooperate in the Regulation of Cathelicidin in Lung Epithelial Cells.Pathogenesis and clinical presentation of rosacea as a key for a symptom-oriented therapy.Pathogenese und Klinik der Rosazea als Schlüssel für eine symptomorientierte Therapie.Evolutionary Origin of the Interferon-Immune Metabolic Axis: The Sterol-Vitamin D Link.Calcium, Orai1, and epidermal proliferation.Vitamin D Status and Tooth Loss in Postmenopausal Females: The Buffalo Osteoporosis and Periodontal Disease (OsteoPerio) Study.Imatinib Triggers Phagolysosome Acidification and Antimicrobial Activity against Mycobacterium bovis Bacille Calmette-Guérin in Glucocorticoid-Treated Human Macrophages.Lowered humidity produces human epidermal equivalents with enhanced barrier properties [Current insights into the pathophysiology of rosacea].Potential Use of 1-25-dihydroxyvitamin D in the Diagnosis and Treatment of Papillary Thyroid Cancer.Transcriptional regulation of cathelicidin genes in chicken bone marrow cells.

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P2860

Regulation of cathelicidin antimicrobial peptide expression by an endoplasmic reticulum (ER) stress signaling, vitamin D receptor-independent pathway

http://www.wikidata.org/entity/Q35311159

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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JBC.M111.250431

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Journal of Biological Chemistry

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Regulation of cathelicidin ant ...... D receptor-independent pathway

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Donald Mackenzie

http://www.w3.org/2001/XMLSchema#string

Kyungho Park

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Peter M Elias

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Theodora Mauro

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Walter M Holleran

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Yoshikazu Uchida

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Yuko Oda

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P2860

Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response

Differentiation-associated expression of ceramidase isoforms in cultured keratinocytes and epidermis

Keratinocyte production of cathelicidin provides direct activity against bacterial skin pathogens

Endoplasmic reticulum stress and the inflammatory basis of metabolic disease

The skin barrier as an innate immune element

Human cathelicidin antimicrobial peptide (CAMP) gene is a direct target of the vitamin D receptor and is strongly up-regulated in myeloid cells by 1,25-dihydroxyvitamin D3

Chemical chaperones reduce ER stress and restore glucose homeostasis in a mouse model of type 2 diabetes

Cell death and endoplasmic reticulum stress: disease relevance and therapeutic opportunities

NF-kappaB-dependent induction of cathelicidin-related antimicrobial peptide in murine mast cells by lipopolysaccharide

CCAAT/enhancer binding protein alpha is a regulatory switch sufficient for induction of granulocytic development from bipotential myeloid progenitors

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21832078

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endoplasmic reticulum

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3190812

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