CaV1.3 channels and intracellular calcium mediate osmotic stress-induced N-terminal c-Jun kinase activation and disruption of tight junctions in Caco-2 CELL MONOLAYERS.
about
Cyclic stretch disrupts apical junctional complexes in Caco-2 cell monolayers by a JNK-2-, c-Src-, and MLCK-dependent mechanism.Ethanol impairs intestinal barrier function in humans through mitogen activated protein kinase signaling: a combined in vivo and in vitro approach.Post-ovulatory aging of oocytes disrupts kinase signaling pathways and lysosome biogenesisJNK is a novel regulator of intercellular adhesionCalcium/Ask1/MKK7/JNK2/c-Src signalling cascade mediates disruption of intestinal epithelial tight junctions by dextran sulfate sodium.A Novel Role of OS-9 in the Maintenance of Intestinal Barrier Function from Hypoxia-induced Injury via p38-dependent Pathway.Modulation of intercellular junctions by cyclic-ADT peptides as a method to reversibly increase blood-brain barrier permeability.Ca2+/Calmodulin and Apo-Calmodulin Both Bind to and Enhance the Tyrosine Kinase Activity of c-SrcEndoplasmic reticulum stress signaling in mammalian oocytes and embryos: life in balanceMouse sperm membrane potential hyperpolarization is necessary and sufficient to prepare sperm for the acrosome reactionInitiation of human astrovirus type 1 infection was blocked by inhibitors of phosphoinositide 3-kinase.The degradation of airway tight junction protein under acidic conditions is probably mediated by transient receptor potential vanilloid 1 receptor.Protection and Restitution of Gut Barrier by Probiotics: Nutritional and Clinical Implications.Calcium Channels and Oxidative Stress Mediate a Synergistic Disruption of Tight Junctions by Ethanol and Acetaldehyde in Caco-2 Cell Monolayers.Ethanol metabolism and its effects on the intestinal epithelial barrier.Transient receptor potential A1 channels regulate epithelial cell barriers formed by MDCK cells.Calcium-mediated oxidative stress: a common mechanism in tight junction disruption by different types of cellular stress.Metformin protects against intestinal barrier dysfunction via AMPKα1-dependent inhibition of JNK signalling activation.Phosphorylation hotspot in the C-terminal domain of occludin regulates the dynamics of epithelial junctional complexes.Ethanol disrupts intestinal epithelial tight junction integrity through intracellular calcium-mediated Rho/ROCK activation.
P2860
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P2860
CaV1.3 channels and intracellular calcium mediate osmotic stress-induced N-terminal c-Jun kinase activation and disruption of tight junctions in Caco-2 CELL MONOLAYERS.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@ast
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@en
type
label
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@ast
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@en
prefLabel
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@ast
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@en
P2093
P2860
P356
P1476
CaV1.3 channels and intracellu ...... ons in Caco-2 CELL MONOLAYERS.
@en
P2093
Damodaran Narayanan
Geetha Samak
Jonathan H Jaggar
Radhakrishna Rao
P2860
P304
30232-30243
P356
10.1074/JBC.M111.240358
P407
P577
2011-07-07T00:00:00Z