Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism - Wikidata - awgldk - TriplyDB

Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

http://www.wikidata.org/entity/Q35315250

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Caveolin-1, a stress-related oncotarget, in drug resistance Nitroglycerin tolerance in caveolin-1 deficient mice.Pulmonary hypertension and metabolic syndrome: Possible connection, PPARγ and Caveolin-1 Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.Nox2-dependent glutathionylation of endothelial NOS leads to uncoupled superoxide production and endothelial barrier dysfunction in acute lung injury.PKC-dependent phosphorylation of eNOS at T495 regulates eNOS coupling and endothelial barrier function in response to G+ -toxins.The sexual dimorphism associated with pulmonary hypertension corresponds to a fibrotic phenotype Oxidative stress and nitric oxide signaling related biomarkers in patients with pulmonary hypertension: a case control study.Serine 1179 Phosphorylation of Endothelial Nitric Oxide Synthase Increases Superoxide Generation and Alters Cofactor Regulation.Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.Chronic hypoxia induces right heart failure in caveolin-1-/- mice.Enhanced caveolin-1 expression in smooth muscle cells: Possible prelude to neointima formation.Enzymatic regulation and functional relevance of NOX5 Reactive oxygen and nitrogen species in pulmonary hypertension.Oxidative stress and autonomic dysregulation contribute to the acute time-dependent myocardial depressant effect of ethanol in conscious female rats.Targeting endothelial dysfunction in vascular complications associated with diabetes.Pathogenesis of pulmonary hypertension: a case for caveolin-1 and cell membrane integrity.Reactive Oxygen and Nitrogen Species in the Development of Pulmonary Hypertension.Modulation of NO and ROS production by AdiNOS transduced vascular cells through supplementation with L-Arg and BH4: implications for gene therapy of restenosis.Nitric oxide donor [Ru(terpy)(bdq)NO]3+ induces uncoupling and phosphorylation of endothelial nitric oxide synthase promoting oxidant production.Caveolin-1 and -2 regulate cell motility in castration-resistant prostate cancer

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

http://www.wikidata.org/entity/Q35315250

description

2011 nî lūn-bûn

@nan

2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հուլիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

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AJPHEART.00936.2010

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American Journal of Physiology - Heart and Circulatory Physiology

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Suppression of eNOS-derived superoxide by caveolin-1: a biopterin-dependent mechanism

@en

P2093

Arturo J Cardounel

http://www.w3.org/2001/XMLSchema#string

Chun-an Chen

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Jay L Zweier

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Kanchana Karuppiah

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Lawrence J Druhan

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Travis Smith

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William C Sessa

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P2860

Tetrahydrobiopterin-dependent preservation of nitric oxide-mediated endothelial function in diabetes by targeted transgenic GTP-cyclohydrolase I overexpression

Caveolin-1 regulates transforming growth factor (TGF)-beta/SMAD signaling through an interaction with the TGF-beta type I receptor

Endothelial nitric oxide synthase is regulated by tyrosine phosphorylation and interacts with caveolin-1

Genetic evidence supporting caveolae microdomain regulation of calcium entry in endothelial cells

The ratio between tetrahydrobiopterin and oxidized tetrahydrobiopterin analogues controls superoxide release from endothelial nitric oxide synthase: an EPR spin trapping study

Dissecting the interaction between nitric oxide synthase (NOS) and caveolin. Functional significance of the nos caveolin binding domain in vivo

Oxidation of tetrahydrobiopterin leads to uncoupling of endothelial cell nitric oxide synthase in hypertension

Endothelial-specific expression of caveolin-1 impairs microvascular permeability and angiogenesis.

Pathophysiological implications of insulin resistance on vascular endothelial function.

Resveratrol enhances GLUT-4 translocation to the caveolar lipid raft fractions through AMPK/Akt/eNOS signalling pathway in diabetic myocardium.

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10.1152/AJPHEART.00936.2010

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