Activity-dependent neuronal plasticity following tissue injury and inflammation.
about
Median nerve trauma in a rat model of work-related musculoskeletal disorderWork-related musculoskeletal disorders of the hand and wrist: epidemiology, pathophysiology, and sensorimotor changesMast cells synthesize, store, and release nerve growth factorSpinal cord mechanisms mediating behavioral hyperalgesia induced by neurokinin-1 tachykinin receptor activation in the rostral ventromedial medullaMonoarticular antigen-induced arthritis leads to pronounced bilateral upregulation of the expression of neurokinin 1 and bradykinin 2 receptors in dorsal root ganglion neurons of ratsNeonatal local noxious insult affects gene expression in the spinal dorsal horn of adult ratsSelective up-regulation of NMDA-NR1 receptor expression in myenteric plexus after TNBS induced colitis in rats.Biological Roles of Resolvins and Related Substances in the Resolution of PainEmerging roles of resolvins in the resolution of inflammation and painAlteration in the voltage dependence of NMDA receptor channels in rat dorsal horn neurones following peripheral inflammationAlterations in spinal cord Fos protein expression induced by bladder stimulation following cystitisPathophysiological tissue changes associated with repetitive movement: a review of the evidenceAcute inflammation induces segmental, bilateral, supraspinally mediated opioid release in the rat spinal cord, as measured by mu-opioid receptor internalizationBrief, low frequency stimulation of rat peripheral C-fibres evokes prolonged microglial-induced central sensitization in adults but not in neonates30 years of dynorphins--new insights on their functions in neuropsychiatric diseasesBi-phasic intensity-dependent opioid-mediated neural amplitude changes in the chinchilla cochlea: partial blockade by an N-Methyl-D-Aspartate (NMDA)-receptor antagonistForebrain mechanisms of nociception and pain: analysis through imaging.Persistent changes in spinal cord gene expression after recovery from inflammatory hyperalgesia: a preliminary study on pain memory.The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain conditionSrc, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptors.The postnatal development of spinal sensory processing.Increased spinal prodynorphin gene expression in reinflammation-associated hyperalgesia after neonatal inflammatory insultPersistent pain model reveals sex difference in morphine potency.Dental injury models: experimental tools for understanding neuroinflammatory interactions and polymodal nociceptor functions.Physiological properties of enkephalin-containing neurons in the spinal dorsal horn visualized by expression of green fluorescent protein in BAC transgenic miceInfluence of the NR3A subunit on NMDA receptor functions.Central non-opioid physiological and pathophysiological effects of dynorphin A and related peptides.NMDA Receptors and Colitis: Basic Science and Clinical Implications.Prolonged nociceptive responses to hind paw formalin injection in rats with a spinal cord injuryRole of inflammation in bladder function and interstitial cystitis.Recognizing pain and distress in laboratory animals.The neurobiology of pain: developmental aspects.Neuropathic pain: what do we do with all these theories?Extreme pain from brown recluse spider bites: model for cytokine-driven pain.TRPA1-expressing primary afferents synapse with a morphologically identified subclass of substantia gelatinosa neurons in the adult rat spinal cord.Protein phosphatase 2A regulates central sensitization in the spinal cord of rats following intradermal injection of capsaicinPeripheral noxious stimulation reduces withdrawal threshold to mechanical stimuli after spinal cord injury: role of tumor necrosis factor alpha and apoptosis.Sensory retraining: a cognitive behavioral therapy for altered sensationReduction of carrageenin oedema and the associated c-Fos expression in the rat lumbar spinal cord by nitric oxide synthase inhibitorActivation of presynaptic NMDA receptors coupled to NaV1.8-resistant sodium channel C-fibers causes retrograde mechanical nociceptor sensitization.
P2860
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P2860
Activity-dependent neuronal plasticity following tissue injury and inflammation.
description
1992 nî lūn-bûn
@nan
1992 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի մարտին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@ast
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@en
type
label
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@ast
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@en
prefLabel
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@ast
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@en
P1476
Activity-dependent neuronal plasticity following tissue injury and inflammation.
@en
P2093
P304
P356
10.1016/0166-2236(92)90019-5
P577
1992-03-01T00:00:00Z