Growth differentiation factor 15 (GDF15)-mediated HER2 phosphorylation reduces trastuzumab sensitivity of HER2-overexpressing breast cancer cells.
about
Evasion of anti-growth signaling: A key step in tumorigenesis and potential target for treatment and prophylaxis by natural compounds.Targeting Src family kinases in anti-cancer therapies: turning promise into triumphNAG-1/GDF15 accumulates in the nucleus and modulates transcriptional regulation of the Smad pathwayThe somatostatin analogue octreotide inhibits growth of small intestine neuroendocrine tumour cellsmRNA profiling reveals determinants of trastuzumab efficiency in HER2-positive breast cancerAn autocrine/paracrine circuit of growth differentiation factor (GDF) 15 has a role for maintenance of breast cancer stem-like cells.Src, a potential target for overcoming trastuzumab resistance in HER2-positive breast carcinoma.P38 MAPK contributes to resistance and invasiveness of HER2- overexpressing breast cancerInsulin-like growth factor-1 receptor signaling increases the invasive potential of human epidermal growth factor receptor 2-overexpressing breast cancer cells via Src-focal adhesion kinase and forkhead box protein M1.Resistance to HER2-targeted therapies: a potential role for FOXM1.Growth differentiation factor 15 stimulates rapamycin-sensitive ovarian cancer cell growth and invasion.Serum Levels of Human MIC-1/GDF15 Vary in a Diurnal Pattern, Do Not Display a Profile Suggestive of a Satiety Factor and Are Related to BMI.LncRNA-ATB promotes trastuzumab resistance and invasion-metastasis cascade in breast cancer.Pharmacological strategies to overcome HER2 cross-talk and Trastuzumab resistance.Catalytic deoxyribozyme-modified nanoparticles for RNAi-independent gene regulationMolecular Mechanisms of Trastuzumab-Based Treatment in HER2-Overexpressing Breast CancerMutant GDF15 presents a poor prognostic outcome for patients with oral squamous cell carcinomaQuercetin induces caspase-dependent extrinsic apoptosis through inhibition of signal transducer and activator of transcription 3 signaling in HER2-overexpressing BT-474 breast cancer cells.New developments in the treatment of HER2-positive breast cancerMechanisms of Adipocytokine-Mediated Trastuzumab Resistance in HER2-Positive Breast Cancer Cell Lines.GDF15 regulates Kv2.1-mediated outward K+ current through the Akt/mTOR signalling pathway in rat cerebellar granule cellsDownregulation of growth differentiation factor-15 in trichostatin A-induced apoptosis could play a role in progression of gastric cancer.Growth differentiation factor 15 mediates epithelial mesenchymal transition and invasion of breast cancers through IGF-1R-FoxM1 signaling.Predicting and Overcoming Chemotherapeutic Resistance in Breast Cancer.GDF15 promotes the proliferation of cervical cancer cells by phosphorylating AKT1 and Erk1/2 through the receptor ErbB2.
P2860
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P2860
Growth differentiation factor 15 (GDF15)-mediated HER2 phosphorylation reduces trastuzumab sensitivity of HER2-overexpressing breast cancer cells.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Growth differentiation factor ...... xpressing breast cancer cells.
@ast
Growth differentiation factor ...... xpressing breast cancer cells.
@en
type
label
Growth differentiation factor ...... xpressing breast cancer cells.
@ast
Growth differentiation factor ...... xpressing breast cancer cells.
@en
prefLabel
Growth differentiation factor ...... xpressing breast cancer cells.
@ast
Growth differentiation factor ...... xpressing breast cancer cells.
@en
P2093
P2860
P1476
Growth differentiation factor ...... xpressing breast cancer cells.
@en
P2093
Jayashree P Joshi
Nicole E Brown
Rita Nahta
Samantha E Griner
P2860
P304
P356
10.1016/J.BCP.2011.07.082
P407
P577
2011-07-23T00:00:00Z