Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis. - Wikidata - awgldk - TriplyDB

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

http://www.wikidata.org/entity/Q35328564

about

Anthrax lethal toxin downregulates claudin-5 expression in human endothelial tight junctions Defense at the border: the blood-brain barrier versus bacterial foreigners Bacillus anthracis factors for phagosomal escape Bacillus anthracis edema factor substrate specificity: evidence for new modes of action Cytoskeleton as an emerging target of anthrax toxins Colonic immune suppression, barrier dysfunction, and dysbiosis by gastrointestinal bacillus anthracis Infection.Lethal factor, but not edema factor, is required to cause fatal anthrax in cynomolgus macaques after pulmonary spore challenge Impaired colonic B-cell responses by gastrointestinal Bacillus anthracis infection The role of autophagy during group B Streptococcus infection of blood-brain barrier endothelium B. anthracis associated cardiovascular dysfunction and shock: the potential contribution of both non-toxin and toxin components Raxibacumab augments hemodynamic support and improves outcomes during shock with B. anthracis edema toxin alone or together with lethal toxin in canines.Analysis of defined combinations of monoclonal antibodies in anthrax toxin neutralization assays and their synergistic action.Differential contribution of Bacillus anthracis toxins to pathogenicity in two animal models Vector-mediated selective expression of lethal factor, a toxic element of Bacillus anthracis, damages A549 cells via inhibition of MAPK and AKT pathways.Diminished but Not Abolished Effect of Two His351 Mutants of Anthrax Edema Factor in a Murine Model B. anthracis edema toxin increases cAMP levels and inhibits phenylephrine-stimulated contraction in a rat aortic ring model Cholera toxin disrupts barrier function by inhibiting exocyst-mediated trafficking of host proteins to intestinal cell junctions New insights into the biological effects of anthrax toxins: linking cellular to organismal responses Streptococcus pneumoniae induces pyroptosis through the regulation of autophagy in murine microglia.Pathology of wild-type and toxin-independent Bacillus anthracis meningitis in rabbits.Gastric pH and Toxin Factors Modulate Infectivity and Disease Progression After Gastrointestinal Exposure to Bacillus anthracis.

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Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

http://www.wikidata.org/entity/Q35328564

description

2011 nî lūn-bûn

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2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2011 թվականի ապրիլին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@ast

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@en

type

label

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@ast

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@en

prefLabel

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@ast

Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@en

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Infection and Immunity

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Contribution of lethal toxin and edema toxin to the pathogenesis of anthrax meningitis.

@en

P2093

Celia M Ebrahimi

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Christian W Renken

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Kelly S Doran

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Roberta A Gottlieb

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Tamsin R Sheen

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P2860

LC3, GABARAP and GATE16 localize to autophagosomal membrane depending on form-II formation

Susceptibility of mitogen-activated protein kinase kinase family members to proteolysis by anthrax lethal factor

Contribution of individual toxin components to virulence of Bacillus anthracis

Invasion of brain microvascular endothelial cells by group B streptococci

Listeria monocytogenes invades the epithelial junctions at sites of cell extrusion

Lung epithelial injury by B. anthracis lethal toxin is caused by MKK-dependent loss of cytoskeletal integrity

Autophagy, Immunity, and Microbial Adaptations

Pertussis toxin transiently affects barrier integrity, organelle organization and transmigration of monocytes in a human brain microvascular endothelial cell barrier model

Proteolytic inactivation of MAP-kinase-kinase by anthrax lethal factor

Pneumococcal trafficking across the blood-brain barrier. Molecular analysis of a novel bidirectional pathway

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2510-2518

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