Participation of lipopolysaccharide-binding protein in lipopolysaccharide-dependent macrophage activation.
about
Dual role of lipopolysaccharide (LPS)-binding protein in neutralization of LPS and enhancement of LPS-induced activation of mononuclear cellsBacterial lipopolysaccharide inhibits dengue virus infection of primary human monocytes/macrophages by blockade of virus entry via a CD14-dependent mechanism.Clinical Significance of Molecular Diagnostic Tools for Bacterial Bloodstream Infections: A Systematic ReviewPotential applications of N-terminal recombinant fragments of bactericidal/permeability-increasing protein in liver surgery.Endotoxin-mediated endothelial cell injury and activation: role of soluble CD14.Extracellular accumulation of potently microbicidal bactericidal/permeability-increasing protein and p15s in an evolving sterile rabbit peritoneal inflammatory exudate.New approaches to sepsis: molecular diagnostics and biomarkers.Towards a rational development of anti-endotoxin agents: novel approaches to sequestration of bacterial endotoxins with small molecules.Receptors, mediators, and mechanisms involved in bacterial sepsis and septic shock.Effect of anticoagulants on binding and neutralization of lipopolysaccharide by the peptide immunoglobulin conjugate CAP18(106-138)-immunoglobulin G in whole bloodThe Lung-Liver Axis: A Requirement for Maximal Innate Immunity and Hepatoprotection during Pneumonia.Activation of Hepatic STAT3 Maintains Pulmonary Defense during EndotoxemiaMyd88 deficiency influences murine tracheal epithelial metaplasia and submucosal gland abundance.A critical role for monocytes and CD14 in endotoxin-induced endothelial cell activation.Late Effects of Total-Body Gamma Irradiation on Cardiac Structure and Function in Male Rhesus Macaques.Lipopolysaccharide (LPS)-binding protein accelerates the binding of LPS to CD14.Helicobacter pylori and Porphyromonas gingivalis lipopolysaccharides are poorly transferred to recombinant soluble CD14.The myeloid differentiation antigen CD14 is N- and O-glycosylated. Contribution of N-linked glycosylation to different soluble CD14 isoforms.Immunoreactivity and bioactivity of lipopolysaccharide-binding protein in normal and heat-inactivated sera.Human lipopolysaccharide-binding protein potentiates bactericidal activity of human bactericidal/permeability-increasing protein.Activation of human endothelial cells by viable or heat-killed gram-negative bacteria requires soluble CD14.Brucella abortus as a potential vaccine candidate: induction of interleukin-12 secretion and enhanced B7.1 and B7.2 and intercellular adhesion molecule 1 surface expression in elutriated human monocytes stimulated by heat-inactivated B. abortus.Helicobacter pylori lipopolysaccharide can activate 70Z/3 cells via CD14Neither CD14 nor serum is absolutely necessary for activation of mononuclear phagocytes by bacterial lipopolysaccharide.Lipopolysaccharide induces expression of fibronectin alpha 5 beta 1-integrin receptors in human monocytic cells in a protein kinase C-dependent fashion.Endotoxins, cytokines, and neuroimmune networks with special reference to HIV infection.Characterization of proinflammatory cytokine production and CD14 expression by murine alveolar macrophage cell lines.Effects of anti-CD18 and LPS on CD14 expression on human monocytes.Lipopolysaccharide induces expression of granulocyte/macrophage colony-stimulating factor, interleukin-8, and interleukin-6 in human nasal, but not lung, fibroblasts: evidence for heterogeneity within the respiratory tract.Neutrophils play a critical role in development of LPS-induced airway disease.Neuron-conditioned media differentially affect the survival of activated or unstimulated microglia: evidence for neuronal control on apoptotic elimination of activated microglia.Toll-like receptor 4 antagonist (E5564) prevents the chronic airway response to inhaled lipopolysaccharide.Neurohormonal host defense in endotoxin shock.Secretory cell differentiation and mucus secretion in cultures of human nasal epithelial cells: use of a monoclonal antibody to study human nasal mucin.Lipopolysaccharide-binding protein in microbial invasion of the amniotic cavity and human parturition.Effects of indomethacin, dexamethasone, and erythromycin on endotoxin-induced intraepithelial mucus production of rat nasal epithelium.
P2860
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P2860
Participation of lipopolysaccharide-binding protein in lipopolysaccharide-dependent macrophage activation.
description
1992 nî lūn-bûn
@nan
1992 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Participation of lipopolysacch ...... pendent macrophage activation.
@ast
Participation of lipopolysacch ...... pendent macrophage activation.
@en
type
label
Participation of lipopolysacch ...... pendent macrophage activation.
@ast
Participation of lipopolysacch ...... pendent macrophage activation.
@en
prefLabel
Participation of lipopolysacch ...... pendent macrophage activation.
@ast
Participation of lipopolysacch ...... pendent macrophage activation.
@en
P2093
P2860
P356
P1476
Participation of lipopolysacch ...... pendent macrophage activation.
@en
P2093
Kravchenko V
Mathison J
Ulevitch RJ
P2860
P304
P356
10.1165/AJRCMB/7.3.239
P577
1992-09-01T00:00:00Z