Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction? - Wikidata - awgldk - TriplyDB

Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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In smokers, Sonic hedgehog modulates pulmonary endothelial function through vascular endothelial growth factor.Arginase Inhibitors: A Rational Approach Over One Century.Pulmonary Arterial Hypertension and Endothelial Dysfunction Is Linked to NADPH Oxidase-Derived Superoxide Formation in Venous Thrombosis and Pulmonary Embolism in Mice.

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

http://www.wikidata.org/entity/Q35339312

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2015 թուականի Մարտին հրատարակուած գիտական յօդուած

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2015 թվականի մարտին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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name

Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Respiratory Research

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Is arginase a potential drug target in tobacco-induced pulmonary endothelial dysfunction?

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Christelle Maurey

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Christophe Delclaux

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Philippe Devillier

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Priscilla Henno

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P2860

Role of the arginine-nitric oxide pathway in the regulation of vascular smooth muscle cell proliferation

Inducible NO synthase dependent S-nitrosylation and activation of arginase1 contribute to age-related endothelial dysfunction

Roles of accumulated endogenous nitric oxide synthase inhibitors, enhanced arginase activity, and attenuated nitric oxide synthase activity in endothelial cells for pulmonary hypertension in rats

L-Arginine supplementation or arginase inhibition augments reflex cutaneous vasodilatation in aged human skin.

Up-regulation of arginase activity contributes to attenuated reflex cutaneous vasodilatation in hypertensive humans.

The effect of L-arginine and creatine on vascular function and homocysteine metabolism.

Nitric oxide as a signaling molecule in the vascular system: an overview.

Local arginase inhibition during early reperfusion mediates cardioprotection via increased nitric oxide production.

Dysregulated arginine metabolism, hemolysis-associated pulmonary hypertension, and mortality in sickle cell disease

Arginase 1 contributes to diminished coronary arteriolar dilation in patients with diabetes

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scholarly article

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Françoise Le Pimpec-Barthes

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