Granzyme B-induced mitochondrial ROS are required for apoptosis - Wikidata - awgldk - TriplyDB

Granzyme B-induced mitochondrial ROS are required for apoptosis

Granzyme B-induced mitochondrial ROS are required for apoptosis

http://www.wikidata.org/entity/Q35349538

about

Regulatory T Cells: Molecular Actions on Effector Cells in Immune Regulation Granzyme B enters the mitochondria in a Sam50-, Tim22- and mtHsp70-dependent manner to induce apoptosis.Antifungal Action of Methylene Blue Involves Mitochondrial Dysfunction and Disruption of Redox and Membrane Homeostasis in C. albicans.X-linked inhibitor of apoptosis protein mediates tumor cell resistance to antibody-dependent cellular cytotoxicity.Metabolic pathways regulated by TAp73 in response to oxidative stress Impact of Mediterranean diet on metabolic syndrome, cancer and longevity.Inhibition of cancer antioxidant defense by natural compounds.ER-mitochondria contacts control surface glycan expression and sensitivity to killer lymphocytes in glioma stem-like cells.Nimbolide inhibits pancreatic cancer growth and metastasis through ROS-mediated apoptosis and inhibition of epithelial-to-mesenchymal transition.A High Yield and Cost-efficient Expression System of Human Granzymes in Mammalian Cells.ROS signaling during granzyme B-mediated apoptosis.Granzyme B-Mediated Activation-Induced Death of CD4+ T Cells Inhibits Murine Acute Graft-versus-Host Disease.Decline of cell viability and mitochondrial activity in mouse skeletal muscle cell in a hypomagnetic field.Human regulatory T cells undergo self-inflicted damage via granzyme pathways upon activation.Granzyme B Disrupts Central Metabolism and Protein Synthesis in Bacteria to Promote an Immune Cell Death Program.Metabolic Reprogramming During Multidrug Resistance in Leukemias.The role of the mitochondria and the endoplasmic reticulum contact sites in the development of the immune responses.Elevated levels of granzyme B correlated with miR-874-3p downregulation in patients with acute myocardial infarction.Granzyme B deficiency promotes osteoblastic differentiation and calcification of vascular smooth muscle cells in hypoxic pulmonary hypertension.Alterations of Oxidative Phosphorylation Complexes in Papillary Thyroid Carcinoma.Natural Killer Cells: Development, Maturation, and Clinical Utilization

P2860

Q26746113-BD10CA5B-2287-4C4D-9B59-1A329029FCE3 Q30152650-F5EB4C07-1571-40A9-A707-8ED6AFC91B10 Q36658607-8D9080A7-4D01-4461-9CE9-37E1AB13BA98 Q36751784-531CDF15-BBFB-4813-822A-B3A41F303684 Q37327366-5127A26B-0E38-4F01-A799-BBA65817A4AE Q37702835-F21978FB-A213-4646-B02F-64AD626A3E7D Q37716657-9B6CB6E1-6F76-44D8-9915-CC70A703059F Q38712677-2AF9A9A3-ABA7-4992-89AF-A8FF0B01592E Q38799932-204F125F-C068-41CD-8835-EB80DAF96171 Q41129118-9EFAB915-8E96-4CFF-B1BA-7898CB72FFEB Q41983916-1F5EE887-10D4-419E-A163-21047CAE548B Q43213211-96DA98B5-B082-40CB-B7A5-A27DDA7218E8 Q46575557-927F6B81-C3A5-4FA3-B033-377276C9982D Q47275057-5A3D0548-1D7A-4EF2-B2B6-A85608C7B878 Q47377299-193065D5-CF59-4C7D-89BC-B19C23E5E1A8 Q52575780-8FF3702D-079F-4C93-9C0E-CBE56FC0837C Q52680707-2A766BBF-C0CE-45AF-825B-6D9C55DE72DD Q54007969-F79EF632-882A-40E3-8EFA-0355C6D20FFD Q54968709-43E12F92-AE70-4E0A-9315-77EB44C539E0 Q55401333-3A3FA892-3E5B-44A4-A9E4-3FE1AE5FDA38 Q58785734-544E9E44-5384-48F5-8355-DC80434F078C

P2860

Granzyme B-induced mitochondrial ROS are required for apoptosis

http://www.wikidata.org/entity/Q35349538

description

2014 nî lūn-bûn

@nan

2014 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Granzyme B-induced mitochondrial ROS are required for apoptosis

@ast

Granzyme B-induced mitochondrial ROS are required for apoptosis

@en

type

label

Granzyme B-induced mitochondrial ROS are required for apoptosis

@ast

Granzyme B-induced mitochondrial ROS are required for apoptosis

@en

prefLabel

Granzyme B-induced mitochondrial ROS are required for apoptosis

@ast

Granzyme B-induced mitochondrial ROS are required for apoptosis

@en

P1433

Q35349538-91E6A82D-DB8C-40C4-84C2-B227FF7CBF70

P1476

Q35349538-A3167053-EB99-4464-B5CE-61875AF390D3

P2093

Q35349538-0C1E3F75-C00E-41A0-9BA7-339AF0023B28

Q35349538-1DBF629D-F3DE-4E45-8F31-6DAA6B74F234

Q35349538-1DE07605-967F-4FEB-9707-895D290D2BD4

Q35349538-7D9F8AED-790E-4755-888F-137A2A12A20A

Q35349538-8A8198A0-41E0-47EF-86F9-2377C4FCF476

Q35349538-B1FDEE82-AAEF-4BE6-AAFD-D671717B43B1

Q35349538-CD7182F4-863F-4271-93DE-5CAFC74685F6

P2860

Q35349538-00942D24-7A25-4401-85BB-CEC7E299E91F

Q35349538-01AEC614-591E-46A3-B1CC-BCC64E9371F0

Q35349538-1319CAD2-6FBC-428B-8F8F-C2A2B19C95DF

Q35349538-1949A068-85DB-40D8-94DD-805AD3AB2C82

Q35349538-1CC6DC04-FBF7-4C6B-99CC-2F3687B6A86A

Q35349538-24F7E981-8F08-47C7-9ABB-175ABC2325A0

Q35349538-2B6E0E5E-7EB4-45EA-AB8D-CF76E3FAD14E

Q35349538-304FE916-CDCA-4903-822B-26F26E3B1377

Q35349538-33C4AC64-CB15-4078-8058-E9E6BC186258

Q35349538-3400E705-61BB-47F1-9FFD-90D26F0E2DAE

P2888

Q35349538-FD44BD9B-BE8F-498A-9902-9312840100F9

P304

Q35349538-963D91DC-B5B8-427B-977E-F554D9CC6047

P31

Q35349538-E42E57FA-1606-437C-8855-1793BAA36671

P356

Q35349538-33E1C421-6D4C-443E-AD16-E79C21326A31

P433

Q35349538-0EAD435D-3B75-4A47-8DE8-8FBA97296869

P478

Q35349538-C2F1264C-854D-4944-AAA6-2B8F8A2CDC3A

P50

Q35349538-AD5B6F50-9133-47B2-A418-586F9660B4E6

P577

Q35349538-8A24F574-529C-4E4A-8C82-187C873BB9B4

P5875

Q35349538-7F87607D-DC3F-481A-8F1B-E5DF40F8526C

P698

Q35349538-F45CEBD5-26FC-4BD8-89CB-2FC276037122

P932

Q35349538-FC8A81A8-88D4-45A9-8447-D2F10691D05F

P356

P1433

Cell Death & Differentiation

P1476

Granzyme B-induced mitochondrial ROS are required for apoptosis

@en

P2093

A Kasahara

http://www.w3.org/2001/XMLSchema#string

D Margiotta

http://www.w3.org/2001/XMLSchema#string

D Martinvalet

http://www.w3.org/2001/XMLSchema#string

G Jacquemin

http://www.w3.org/2001/XMLSchema#string

J Lieberman

http://www.w3.org/2001/XMLSchema#string

J Thiery

http://www.w3.org/2001/XMLSchema#string

M Walch

http://www.w3.org/2001/XMLSchema#string

P2860

Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

NDUFA4 is a subunit of complex IV of the mammalian electron transport chain

How mitochondria produce reactive oxygen species

The major human and mouse granzymes are structurally and functionally divergent

Analysis of the assembly profiles for mitochondrial- and nuclear-DNA-encoded subunits into complex I

Tumor suppressor NM23-H1 is a granzyme A-activated DNase during CTL-mediated apoptosis, and the nucleosome assembly protein SET is its inhibitor

The nuclear encoded subunits of complex I from bovine heart mitochondria

OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion

Granzyme B directly and efficiently cleaves several downstream caspase substrates: implications for CTL-induced apoptosis

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

P2888

P304

862-874

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/CDD.2014.180

http://www.w3.org/2001/XMLSchema#string

P433

5

http://www.w3.org/2001/XMLSchema#string

P478

22

http://www.w3.org/2001/XMLSchema#string

P50

Esen Yonca Bassoy

P577

2014-10-31T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

267746061

http://www.w3.org/2001/XMLSchema#string

P698

25361078

http://www.w3.org/2001/XMLSchema#string

P932

4392081

http://www.w3.org/2001/XMLSchema#string