miR-222 is necessary for exercise-induced cardiac growth and protects against pathological cardiac remodeling. - Wikidata - awgldk - TriplyDB

miR-222 is necessary for exercise-induced cardiac growth and protects against pathological cardiac remodeling.

miR-222 is necessary for exercise-induced cardiac growth and protects against pathological cardiac remodeling.

http://www.wikidata.org/entity/Q35372100

about

Non-coding RNAs in cardiac regeneration Exercise for the heart: signaling pathways Aerobic exercise training promotes physiological cardiac remodeling involving a set of microRNAs The Emerging Role of MicroRNA-155 in Cardiovascular Diseases The Role of Exercise in Cardiac Aging: From Physiology to Molecular Mechanisms miR-222 in Exercise-Induced Cardiac Growth MicroRNA Expression Signature in Human Calcific Aortic Valve Disease.Mir30c Is Involved in Diabetic Cardiomyopathy through Regulation of Cardiac Autophagy via BECN1 Differential expression of microRNAs following cardiopulmonary bypass in children with congenital heart diseases Circulating miR-30d Predicts Survival in Patients with Acute Heart Failure.Identification of General and Heart-Specific miRNAs in Sheep (Ovis aries)MicroRNA-222 Controls Human Pancreatic Cancer Cell Line Capan-2 Proliferation by P57 Targeting.Influence of Physical Activity on Hypertension and Cardiac Structure and Function Transgenic Expression of miR-222 Disrupts Intestinal Epithelial Regeneration by Targeting Multiple Genes Including Frizzled-7.Telocytes in exercise-induced cardiac growth miR-19b attenuates H2O2-induced apoptosis in rat H9C2 cardiomyocytes via targeting PTEN Circulating miR-21, miR-378, and miR-940 increase in response to an acute exhaustive exercise in chronic heart failure patients CITED4 induces physiologic hypertrophy and promotes functional recovery after ischemic injury.ANGPTL2 activity in cardiac pathologies accelerates heart failure by perturbing cardiac function and energy metabolism.Exercise mediated protection of diabetic heart through modulation of microRNA mediated molecular pathways MiR-222 in Cardiovascular Diseases: Physiology and Pathology miR-17-3p Contributes to Exercise-Induced Cardiac Growth and Protects against Myocardial Ischemia-Reperfusion Injury.Bioinformatics Analysis Reveals MicroRNAs Regulating Biological Pathways in Exercise-Induced Cardiac Physiological Hypertrophy.MiR-30c protects diabetic nephropathy by suppressing epithelial-to-mesenchymal transition in db/db mice.Exercise-induced circulating extracellular vesicles protect against cardiac ischemia-reperfusion injury.Exercise and epigenetic inheritance of disease risk.How cardiomyocytes sense pathophysiological stresses for cardiac remodeling.Redirecting cardiac growth mechanisms for therapeutic regeneration.Decoding Telocytes.The Role of MicroRNAs in the Cardiac Response to Exercise.Molecular Mechanisms Underlying Cardiac Adaptation to Exercise.miR-199-sponge transgenic mice develop physiological cardiac hypertrophy.Circulating exosome microRNA associated with heart failure secondary to myxomatous mitral valve disease in a naturally occurring canine model.Cardiac cell proliferation is not necessary for exercise-induced cardiac growth but required for its protection against ischaemia/reperfusion injury.BET bromodomain inhibition suppresses innate inflammatory and profibrotic transcriptional networks in heart failure.Bone marrow-derived cells and their conditioned medium induce microvascular repair in uremic rats by stimulation of endogenous repair mechanisms.MicroRNA Expression Signature in Degenerative Aortic Stenosis.miR-19b controls cardiac fibroblast proliferation and migration.miRNAs and sports: tracking training status and potentially confounding diagnoses Exercise for cardiac health and regeneration: killing two birds with one stone.

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P2860

miR-222 is necessary for exercise-induced cardiac growth and protects against pathological cardiac remodeling.

http://www.wikidata.org/entity/Q35372100

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

miR-222 is necessary for exerc ...... thological cardiac remodeling.

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miR-222 is necessary for exerc ...... thological cardiac remodeling.

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type

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miR-222 is necessary for exerc ...... thological cardiac remodeling.

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miR-222 is necessary for exerc ...... thological cardiac remodeling.

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miR-222 is necessary for exerc ...... thological cardiac remodeling.

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miR-222 is necessary for exerc ...... thological cardiac remodeling.

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J.CMET.2015.02.014

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Cell Metabolism

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miR-222 is necessary for exerc ...... thological cardiac remodeling.

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P2093

Anthony Rosenzweig

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Chunxiang Zhang

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Chunyang Xiao

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Colin Platt

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Federico Damilano

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Han Zhu

http://www.w3.org/2001/XMLSchema#string

Junjie Xiao

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Lin Che

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Pontus Boström

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Vassilios Bezzerides

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P2860

Tumor necrosis factor alpha-induced desumoylation and cytoplasmic translocation of homeodomain-interacting protein kinase 1 are critical for apoptosis signal-regulating kinase 1-JNK/p38 activation

Exercise enhances learning and hippocampal neurogenesis in aged mice.

Evidence for cardiomyocyte renewal in humans

MiR-221 and MiR-222 alterations in sporadic ovarian carcinoma: Relationship to CDKN1B, CDKNIC and overall survival

A necessary role of miR-221 and miR-222 in vascular smooth muscle cell proliferation and neointimal hyperplasia

Regulation of the p27(Kip1) tumor suppressor by miR-221 and miR-222 promotes cancer cell proliferation

A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990-2010: a systematic analysis for the Global Burden of Disease Study 2010.

Neuregulin1/ErbB4 signaling induces cardiomyocyte proliferation and repair of heart injury

Overlapping roles for homeodomain-interacting protein kinases hipk1 and hipk2 in the mediation of cell growth in response to morphogenetic and genotoxic signals.

A cardiac microRNA governs systemic energy homeostasis by regulation of MED13

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4

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21

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Bruce Spiegelman

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25863248

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