Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice. - Wikidata - awgldk - TriplyDB

Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice.

Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice.

http://www.wikidata.org/entity/Q35393618

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Mechanisms of tissue injury in lupus nephritis Therapeutic modulators of STAT signalling for human diseases The inflammasome and lupus: another innate immune mechanism contributing to disease pathogenesis?Biomarker profiling for lupus nephritis.Autocrine CSF-1 and CSF-1 receptor coexpression promotes renal cell carcinoma growth.Targeting the inflammasome in rheumatic diseases.Deletion of IL-18 Expression Ameliorates Spontaneous Kidney Failure in MRLlpr Mice Granulocyte colony-stimulating factor treatment ameliorates lupus nephritis through the expansion of regulatory T cells.Contribution of the STAT4 rs7574865 gene polymorphism to the susceptibility to autoimmune thyroiditis in healthy Turk population and psoriatic subgroups.Univariation and multiple linear regression analyses for 23 single nucleotide polymorphisms in 14 chronic glomerular disease's predisposing genes and systemic lupus erythematosus in Han Chinese.Making sense of therapeutics using antisense technology.The role of cytokines in the pathogenesis of systemic lupus erythematosus - from bench to bedside.Therapeutic potential of STAT4 in autoimmunity.Renal tubular epithelial cell-derived BAFF expression mediates kidney damage and correlates with activity of proliferative lupus nephritis in mouse and men.The Role of STAT Signaling Pathways in the Pathogenesis of Systemic Lupus Erythematosus.Re-Examining Neutrophil Participation in GN.Definition of IFN-γ-related pathways critical for chemically-induced systemic autoimmunity Intrarenal macrophage infiltration induced by T cells is associated with podocyte injury in lupus nephritis patients.

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Targeting transcription factor Stat4 uncovers a role for interleukin-18 in the pathogenesis of severe lupus nephritis in mice.

http://www.wikidata.org/entity/Q35393618

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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name

Targeting transcription factor ...... evere lupus nephritis in mice.

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Targeting transcription factor ...... evere lupus nephritis in mice.

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Targeting transcription factor ...... evere lupus nephritis in mice.

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Targeting transcription factor ...... evere lupus nephritis in mice.

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Targeting transcription factor ...... evere lupus nephritis in mice.

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Targeting transcription factor ...... evere lupus nephritis in mice.

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Kidney International

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Andreas Schwarting

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Birte Kutska

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Julia Menke

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Katelyn T Byrne

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Manfred Relle

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Michaela Blanfeld

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Tillmann Bork

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Vicki R Kelley

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P2860

Stat4, a novel gamma interferon activation site-binding protein expressed in early myeloid differentiation

Interleukin 12 induces tyrosine phosphorylation and activation of STAT4 in human lymphocytes

Stat3 and Stat4: members of the family of signal transducers and activators of transcription

Specificity of the STAT4 genetic association for severe disease manifestations of systemic lupus erythematosus

Phase III study of gemcitabine and cisplatin with or without aprinocarsen, a protein kinase C-alpha antisense oligonucleotide, in patients with advanced-stage non-small-cell lung cancer.

Safe and efficient generation of recombinant retroviruses with amphotropic and ecotropic host ranges

Regulation of the IL-12/IL-12R axis: a critical step in T-helper cell differentiation and effector function.

Monocyte chemoattractant protein-1 promotes macrophage-mediated tubular injury, but not glomerular injury, in nephrotoxic serum nephritis

Rheumatic syndromes associated with complement deficiency.

Spontaneous murine lupus-like syndromes. Clinical and immunopathological manifestations in several strains

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10.1038/KI.2010.438

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