IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
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The Role of Interleukin-22 and Its Receptor in the Development and Pathogenesis of Experimental Autoimmune UveitisThe pathogenic role of interleukin-22 and its receptor during UVB-induced skin inflammation.Increased expression of IL-22 is associated with disease activity in Behcet's disease.Endogenous IL-22 plays a dual role in arthritis: regulation of established arthritis via IFN-γ responses.Dental follicle mesenchymal stem cell administration ameliorates muscle weakness in MuSK-immunized mice.AAV-IL-22 modifies liver chemokine activity and ameliorates portal inflammation in murine autoimmune cholangitis.Interleukin-22 reduces the severity of collagen-induced arthritis in association with increased levels of interleukin-10.Emerging role of interleukin-22 in autoimmune diseasesHealing of intestinal inflammation by IL-22.IL-22, not simply a Th17 cytokine.Pivotal roles of T-helper 17-related cytokines, IL-17, IL-22, and IL-23, in inflammatory diseases.The role of Th17-associated cytokines in the pathogenesis of experimental autoimmune uveitis (EAU).The parasitic worm product ES-62 up-regulates IL-22 production by γδ T cells in the murine model of Collagen-Induced Arthritis.Abrogation of Rbpj attenuates experimental autoimmune uveoretinitis by inhibiting IL-22-producing CD4+ T cells.Long-term therapeutic effects of mesenchymal stem cells compared to dexamethasone on recurrent experimental autoimmune uveitis of rats.The multiple sclerosis risk gene IL22RA2 contributes to a more severe murine autoimmune neuroinflammation.Interleukin 17A as an effective target for anti-inflammatory and antiparasitic treatment of toxoplasmic uveitis.Expression of interleukin-22 in myasthenia gravis.Serum cytokine profile in adalimumab-treated refractory uveitis patients: decreased IL-22 correlates with clinical responses.Pharmacological Inhibition of Bromodomain Proteins Suppresses Retinal Inflammatory Disease and Downregulates Retinal Th17 Cells.Regulation of IL-22BP in psoriasis.Immune Privilege and Eye-Derived T-Regulatory Cells.IL-22 deficiency in donor T cells attenuates murine acute graft-versus-host disease mortality while sparing the graft-versus-leukemia effectAnti-IL-22 Antibody Attenuates Acute Graft-versus-Host Disease via Increasing Foxp3 T Cell through Modulation of CD11b Cell Function
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P2860
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@ast
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@en
type
label
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@ast
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@en
prefLabel
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@ast
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@en
P2093
P2860
P356
P1476
IL-22-induced regulatory CD11b+ APCs suppress experimental autoimmune uveitis.
@en
P2093
Deming Sun
Guomin Jiang
Henry J Kaplan
P2860
P304
P356
10.4049/JIMMUNOL.1100482
P407
P577
2011-07-27T00:00:00Z