Cutting edge: regulatory T cells do not mediate suppression via programmed cell death pathways - Wikidata - awgldk - TriplyDB

Cutting edge: regulatory T cells do not mediate suppression via programmed cell death pathways

Cutting edge: regulatory T cells do not mediate suppression via programmed cell death pathways

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Necroptosis in tumorigenesis, activation of anti-tumor immunity, and cancer therapy Regulation generation: the suppressive functions of human regulatory T cells Defective cell death signalling along the Bcl-2 regulated apoptosis pathway compromises Treg cell development and limits their functionality in mice Mechanisms of T(reg) Suppression: Still a Long Way to Go.The BH3-only proteins Bim and Puma cooperate to impose deletional tolerance of organ-specific antigens.Molecular mechanisms of treg-mediated T cell suppression Targeting regulatory T cells in tumors.Cytotoxic response of human regulatory T cells upon T-cell receptor-mediated activation: a matter of purity Comment on "Cutting edge: regulatory T cells do not mediate suppression via programmed cell death pathways".

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Cutting edge: regulatory T cells do not mediate suppression via programmed cell death pathways

http://www.wikidata.org/entity/Q35403062

description

2011 nî lūn-bûn

@nan

2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Cutting edge: regulatory T cel ...... programmed cell death pathways

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Cutting edge: regulatory T cel ...... programmed cell death pathways

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type

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Cutting edge: regulatory T cel ...... programmed cell death pathways

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Cutting edge: regulatory T cel ...... programmed cell death pathways

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prefLabel

Cutting edge: regulatory T cel ...... programmed cell death pathways

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Cutting edge: regulatory T cel ...... programmed cell death pathways

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JIMMUNOL.1100548

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Journal of Immunology

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Cutting edge: regulatory T cel ...... programmed cell death pathways

@en

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Andrea L Szymczak-Workman

http://www.w3.org/2001/XMLSchema#string

Dario A A Vignali

http://www.w3.org/2001/XMLSchema#string

Greg M Delgoffe

http://www.w3.org/2001/XMLSchema#string

P2860

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

The NF-kappaB regulator Bcl-3 and the BH3-only proteins Bim and Puma control the death of activated T cells

FOXO3a-dependent regulation of Puma in response to cytokine/growth factor withdrawal

Puma cooperates with Bim, the rate-limiting BH3-only protein in cell death during lymphocyte development, in apoptosis induction

Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

p53- and drug-induced apoptotic responses mediated by BH3-only proteins puma and noxa

Puma is an essential mediator of p53-dependent and -independent apoptotic pathways

The inhibitory cytokine IL-35 contributes to regulatory T-cell function

RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis

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4416-4420

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scholarly article

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10.4049/JIMMUNOL.1100548

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9

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187

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Douglas R. Green

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2011-09-26T00:00:00Z

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51676059

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21949016

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3197948

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